Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice.
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The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesisSevere acute respiratory syndrome coronavirus 7a accessory protein is a viral structural proteinRewiring the severe acute respiratory syndrome coronavirus (SARS-CoV) transcription circuit: engineering a recombination-resistant genomeCoronavirus virulence genes with main focus on SARS-CoV envelope geneCoronavirus gene 7 counteracts host defenses and modulates virus virulenceGenome-wide analysis of protein-protein interactions and involvement of viral proteins in SARS-CoV replicationAmino acids 15-28 in the ectodomain of SARS coronavirus 3a protein induces neutralizing antibodies.A comprehensive collection of systems biology data characterizing the host response to viral infection.Bat Severe Acute Respiratory Syndrome-Like Coronavirus WIV1 Encodes an Extra Accessory Protein, ORFX, Involved in Modulation of the Host Immune Response.The open reading frame 3a protein of severe acute respiratory syndrome-associated coronavirus promotes membrane rearrangement and cell death.A network integration approach to predict conserved regulators related to pathogenicity of influenza and SARS-CoV respiratory viruses.A mouse-adapted SARS-coronavirus causes disease and mortality in BALB/c mice.Analysis of intraviral protein-protein interactions of the SARS coronavirus ORFeome.MyD88 is required for protection from lethal infection with a mouse-adapted SARS-CoV.Differential stepwise evolution of SARS coronavirus functional proteins in different host speciesThe replicase gene of avian coronavirus infectious bronchitis virus is a determinant of pathogenicityInfidelity of SARS-CoV Nsp14-exonuclease mutant virus replication is revealed by complete genome sequencing.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.Coronaviruses post-SARS: update on replication and pathogenesis.Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.The N-terminal region of severe acute respiratory syndrome coronavirus protein 6 induces membrane rearrangement and enhances virus replication.SARS-CoV 9b protein diffuses into nucleus, undergoes active Crm1 mediated nucleocytoplasmic export and triggers apoptosis when retained in the nucleus.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Porcine epidemic diarrhoea virus: a comprehensive review of molecular epidemiology, diagnosis, and vaccines.A putative diacidic motif in the SARS-CoV ORF6 protein influences its subcellular localization and suppression of expression of co-transfected expression constructs.SARS coronavirus 3b accessory protein modulates transcriptional activity of RUNX1b.Development and characterization of a reverse genetic system for studying dengue virus serotype 3 strain variation and neutralization.Successful vaccination strategies that protect aged mice from lethal challenge from influenza virus and heterologous severe acute respiratory syndrome coronavirus.Infectious Bronchitis Virus as a Vector for the Expression of Heterologous Genes.An immunosuppressed Syrian golden hamster model for SARS-CoV infection.Construction of a severe acute respiratory syndrome coronavirus infectious cDNA clone and a replicon to study coronavirus RNA synthesis.Severe acute respiratory syndrome coronavirus protein 6 mediates ubiquitin-dependent proteosomal degradation of N-Myc (and STAT) interactor.Severe acute respiratory syndrome coronavirus protein 6 accelerates murine coronavirus infections.A severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.Genome Wide Identification of SARS-CoV Susceptibility Loci Using the Collaborative Cross.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionInduction of apoptosis by the severe acute respiratory syndrome coronavirus 7a protein is dependent on its interaction with the Bcl-XL protein.Severe acute respiratory syndrome coronavirus accessory protein 6 is a virion-associated protein and is released from 6 protein-expressing cells.Potent cross-reactive neutralization of SARS coronavirus isolates by human monoclonal antibodies.The Nucleocapsid Protein of Coronaviruses Acts as a Viral Suppressor of RNA Silencing in Mammalian Cells
P2860
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P2860
Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice.
description
2005 nî lūn-bûn
@nan
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@ast
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@en
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@nl
type
label
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@ast
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@en
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@nl
prefLabel
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@ast
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@en
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@nl
P2093
P2860
P1433
P1476
Severe acute respiratory syndr ...... ion in cell cultures and mice.
@en
P2093
Amy C Sims
Boyd Yount
Damon Deming
Jennifer Sparks
Matthew B Frieman
Nancy Davis
Rhonda S Roberts
P2860
P304
14909-14922
P356
10.1128/JVI.79.23.14909-14922.2005
P407
P577
2005-12-01T00:00:00Z