A severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.
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Development of next-generation respiratory virus vaccines through targeted modifications to viral immunomodulatory genesRelevance of Viroporin Ion Channel Activity on Viral Replication and PathogenesisCoronavirus virulence genes with main focus on SARS-CoV envelope geneCoronavirus reverse genetic systems: infectious clones and repliconsCoronavirus gene 7 counteracts host defenses and modulates virus virulenceGenome-wide analysis of protein-protein interactions and involvement of viral proteins in SARS-CoV replicationStructure and inhibition of the SARS coronavirus envelope protein ion channelA decade after SARS: strategies for controlling emerging coronavirusesStructure of a conserved Golgi complex-targeting signal in coronavirus envelope proteinsThe M, E, and N structural proteins of the severe acute respiratory syndrome coronavirus are required for efficient assembly, trafficking, and release of virus-like particles.Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesisSARS-CoV envelope protein palmitoylation or nucleocapid association is not required for promoting virus-like particle productionPathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.Coronaviruses post-SARS: update on replication and pathogenesis.Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.Coronavirus infection, ER stress, apoptosis and innate immunityEquine torovirus (BEV) induces caspase-mediated apoptosis in infected cellsRNA virus reverse genetics and vaccine design.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Palmitoylation of SARS-CoV S protein is necessary for partitioning into detergent-resistant membranes and cell-cell fusion but not interaction with M protein.The PDZ-binding motif of severe acute respiratory syndrome coronavirus envelope protein is a determinant of viral pathogenesis.Effects of Toll-like receptor stimulation on eosinophilic infiltration in lungs of BALB/c mice immunized with UV-inactivated severe acute respiratory syndrome-related coronavirus vaccine.Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.Culturing the unculturable: human coronavirus HKU1 infects, replicates, and produces progeny virions in human ciliated airway epithelial cell cultures.Virus-specific memory CD8 T cells provide substantial protection from lethal severe acute respiratory syndrome coronavirus infection.A single polar residue and distinct membrane topologies impact the function of the infectious bronchitis coronavirus E protein.The SARS coronavirus E protein interacts with PALS1 and alters tight junction formation and epithelial morphogenesis.Evolved variants of the membrane protein can partially replace the envelope protein in murine coronavirus assembly.Coronaviruses: an overview of their replication and pathogenesis.The hydrophobic domain of infectious bronchitis virus E protein alters the host secretory pathway and is important for release of infectious virus.Identification of a Golgi complex-targeting signal in the cytoplasmic tail of the severe acute respiratory syndrome coronavirus envelope protein.Severe acute respiratory syndrome coronaviruses with mutations in the E protein are attenuated and promising vaccine candidates.Incorporation of spike and membrane glycoproteins into coronavirus virions.The structure and functions of coronavirus genomic 3' and 5' ends.Role of the coronavirus E viroporin protein transmembrane domain in virus assembly.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionA Coronavirus E Protein Is Present in Two Distinct Pools with Different Effects on Assembly and the Secretory Pathway.Coronavirus envelope (E) protein remains at the site of assemblySevere acute respiratory syndrome coronavirus E protein transports calcium ions and activates the NLRP3 inflammasomeCoronavirus E protein forms ion channels with functionally and structurally-involved membrane lipids.
P2860
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P2860
A severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@ast
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@en
type
label
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@ast
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@en
prefLabel
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@ast
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@en
P2093
P2860
P50
P356
P1433
P1476
A severe acute respiratory syn ...... tenuated in vitro and in vivo.
@en
P2093
Elaine Lamirande
Enrique Alvarez
Fernando Almazán
Marta L DeDiego
María Teresa Rejas
Wun-Ju Shieh
P2860
P304
P356
10.1128/JVI.01467-06
P407
P577
2006-11-15T00:00:00Z