DNA-damage response during mitosis induces whole-chromosome missegregation
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Maintaining Genome Stability in Defiance of Mitotic DNA DamageQuantitative assessment of chromosome instability induced through chemical disruption of mitotic progressionDNA damage response during mouse oocyte maturation.Aurora A Kinase Amplifies a Midzone Phosphorylation Gradient to Promote High-Fidelity Cytokinesis.Aneuploidy and chromosomal instability in cancer: a jackpot to chaos.Numerical chromosomal instability mediates susceptibility to radiation treatment.ATM-dependent Phosphorylation of the Fanconi Anemia Protein PALB2 Promotes the DNA Damage Response.DNA damage induces a meiotic arrest in mouse oocytes mediated by the spindle assembly checkpoint.Micronucleus formation causes perpetual unilateral chromosome inheritance in mouse embryos.Rare variants in BRCA2 and CHEK2 are associated with the risk of urinary tract cancers.Important molecular genetic markers of colorectal cancer.Genomic instability: Crossing pathways at the origin of structural and numerical chromosome changes.The Elephant in the Room: The Role of Microtubules in Cancer.Late mitotic functions of Aurora kinases.Centrosome age regulates kinetochore-microtubule stability and biases chromosome mis-segregationFrequently mutated genes/pathways and genomic instability as prevention targets in liver cancer.Living in CIN: Mitotic Infidelity and Its Consequences for Tumor Promotion and Suppression.Chromosomal Instability as a Driver of Tumor Heterogeneity and Evolution.Mitotic Dysfunction Associated with Aging Hallmarks.Mitotic DNA Damage Response: At the Crossroads of Structural and Numerical Cancer Chromosome Instabilities.Dynein prevents erroneous kinetochore-microtubule attachments in mitosis.Mitotic entry: The interplay between Cdk1, Plk1 and Bora.Regulation of chromosome segregation in oocytes and the cellular basis for female meiotic errors.DNA damage induces a kinetochore-based ATM/ATR-independent SAC arrest unique to the first meiotic division in mouse oocytes.Chromosome passenger complex is required for the survival of cells with ring chromosomes in fission yeast.Whole chromosome loss and associated breakage-fusion-bridge cycles transform mouse tetraploid cells.Imprinted survival genes preclude loss of heterozygosity of chromosome 7 in cancer cells.Near-tetraploid cancer cells show chromosome instability triggered by replication stress and exhibit enhanced invasiveness.Genomic Instability Is Induced by Persistent Proliferation of Cells Undergoing Epithelial-to-Mesenchymal Transition.A Distinct Class of Chromoanagenesis Events Characterized by Focal Copy Number Gains.
P2860
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P2860
DNA-damage response during mitosis induces whole-chromosome missegregation
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2014 nî lūn-bûn
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2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
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2014 թվականի օգոստոսին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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DNA-damage response during mitosis induces whole-chromosome missegregation
@ast
DNA-damage response during mitosis induces whole-chromosome missegregation
@en
DNA-damage response during mitosis induces whole-chromosome missegregation
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DNA-damage response during mitosis induces whole-chromosome missegregation
@ast
DNA-damage response during mitosis induces whole-chromosome missegregation
@en
DNA-damage response during mitosis induces whole-chromosome missegregation
@nl
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DNA-damage response during mitosis induces whole-chromosome missegregation
@ast
DNA-damage response during mitosis induces whole-chromosome missegregation
@en
DNA-damage response during mitosis induces whole-chromosome missegregation
@nl
P2093
P2860
P1433
P1476
DNA-damage response during mitosis induces whole-chromosome missegregation
@en
P2093
Bassem I Zaki
John P Murnane
Lilian Kabeche
P2860
P304
P356
10.1158/2159-8290.CD-14-0403
P577
2014-08-08T00:00:00Z