Ascorbic acid regulates osterix expression in osteoblasts by activation of prolyl hydroxylase and ubiquitination-mediated proteosomal degradation pathway
about
Targeted disruption of leucine-rich repeat kinase 1 but not leucine-rich repeat kinase 2 in mice causes severe osteopetrosisInvolvement of Prolyl Hydroxylase Domain Protein in the Rosiglitazone-Induced Suppression of Osteoblast DifferentiationTargeted disruption of ephrin B1 in cells of myeloid lineage increases osteoclast differentiation and bone resorption in mice.Rab GTPase mediated procollagen trafficking in ascorbic acid stimulated osteoblasts.Transgenic overexpression of ephrin b1 in bone cells promotes bone formation and an anabolic response to mechanical loading in mice.Differential expression of claudin family members during osteoblast and osteoclast differentiation: Cldn-1 is a novel positive regulator of osteoblastogenesis.Epiphyseal chondrocyte secondary ossification centers require thyroid hormone activation of Indian hedgehog and osterix signalingConditional Deletion of Prolyl Hydroxylase Domain-Containing Protein 2 (Phd2) Gene Reveals Its Essential Role in Chondrocyte Function and Endochondral Bone Formation.Disruption of claudin-18 diminishes ovariectomy-induced bone loss in miceThe Roles and Mechanisms of Actions of Vitamin C in Bone: New Developments.Thyroid hormone receptor-β1 signaling is critically involved in regulating secondary ossification via promoting transcription of the Ihh gene in the epiphysis.Genetic and molecular control of osterix in skeletal formationThe roles of AKR1C1 and AKR1C2 in ethyl-3,4-dihydroxybenzoate induced esophageal squamous cell carcinoma cell deathProlyl Hydroxylase Domain-Containing Protein 2 (Phd2) Regulates Chondrocyte Differentiation and Secondary Ossification in Mice.Conditional Deletion of the Phd2 Gene in Articular Chondrocytes Accelerates Differentiation and Reduces Articular Cartilage ThicknessRE1-Silencing Transcription Factor (Rest) is a Novel Regulator of Osteoblast Differentiation.NHERF1 regulation of PTH-dependent bimodal Pi transport in osteoblasts.Conditional disruption of the prolyl hydroxylase domain-containing protein 2 (Phd2) gene defines its key role in skeletal development.Small molecules modified biomimetic gelatin/hydroxyapatite nanofibers constructing an ideal osteogenic microenvironment with significantly enhanced cranial bone formation
P2860
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P2860
Ascorbic acid regulates osterix expression in osteoblasts by activation of prolyl hydroxylase and ubiquitination-mediated proteosomal degradation pathway
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@ast
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@en
type
label
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@ast
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@en
prefLabel
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@ast
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@en
P2093
P2860
P1476
Ascorbic acid regulates osteri ...... roteosomal degradation pathway
@en
P2093
Sheila Pourteymoor
Subburaman Mohan
Weirong Xing
P2860
P304
P356
10.1152/PHYSIOLGENOMICS.00229.2010
P577
2011-04-05T00:00:00Z