Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
about
Natural pathogens of laboratory mice, rats, and rabbits and their effects on research.Complete sequence and genomic analysis of murine gammaherpesvirus 68MCMV induces neointima in IFN-gammaR-/- mice: intimal cell apoptosis and persistent proliferation of myofibroblastsInhibition of NF-kappaB activation in vivo impairs establishment of gammaherpesvirus latency.Identification of closely spaced but distinct transcription initiation sites for the murine gammaherpesvirus 68 latency-associated M2 gene.Focal transcriptional activity of murine cytomegalovirus during latency in the lungs.Spliced mRNA encoding the murine cytomegalovirus chemokine homolog predicts a beta chemokine of novel structureIn vivo replication, latency, and immunogenicity of murine cytomegalovirus mutants with deletions in the M83 and M84 genes, the putative homologs of human cytomegalovirus pp65 (UL83).Three distinct regions of the murine gammaherpesvirus 68 genome are transcriptionally active in latently infected mice.Ex vivo stimulation of B cells latently infected with gammaherpesvirus 68 triggers reactivation from latencyBiphasic recruitment of transcriptional repressors to the murine cytomegalovirus major immediate-early promoter during the course of infection in vivoMucosal and parenteral vaccination against acute and latent murine cytomegalovirus (MCMV) infection by using an attenuated MCMV mutant.The murine gammaherpesvirus 68 M2 gene is required for efficient reactivation from latently infected B cells.Elimination of ie1 significantly attenuates murine cytomegalovirus virulence but does not alter replicative capacity in cell cultureConstruction and characterization of murine cytomegaloviruses that contain transposon insertions at open reading frames m09 and M83.In vitro and in vivo characterization of a murine cytomegalovirus with a transposon insertional mutation at open reading frame M43.Murine cytomegalovirus containing a mutation at open reading frame M37 is severely attenuated in growth and virulence in vivo.Establishment and maintenance of long-term murine gammaherpesvirus 68 latency in B cells in the absence of CD40.Murine cytomegalovirus open reading frame M27 plays an important role in growth and virulence in mice.Random, asynchronous, and asymmetric transcriptional activity of enhancer-flanking major immediate-early genes ie1/3 and ie2 during murine cytomegalovirus latency in the lungs.Role of B-cell proliferation in the establishment of gammaherpesvirus latency.Age-related dysregulation of CD8+ T cell memory specific for a persistent virus is independent of viral replicationInflation and long-term maintenance of CD8 T cells responding to a latent herpesvirus depend upon establishment of latency and presence of viral antigens.Reactivation of latent cytomegalovirus infection in mouse brain cells detected after transfer to brain slice cultures.Persisting murine cytomegalovirus can reactivate and has unique transcriptional activity in ocular tissue.Murine cytomegalovirus with a transposon insertional mutation at open reading frame m155 is deficient in growth and virulence in mice.An essential role of the enhancer for murine cytomegalovirus in vivo growth and pathogenesis.In vitro and in vivo characterization of a murine cytomegalovirus with a mutation at open reading frame m166.Transmission of murine cytomegalovirus in breast milk: a model of natural infection in neonatesMurine cytomegalovirus with a transposon insertional mutation at open reading frame M35 is defective in growth in vivo.Long-term latent murine Gammaherpesvirus 68 infection is preferentially found within the surface immunoglobulin D-negative subset of splenic B cells in vivo.Transplacental murine cytomegalovirus infection in the brain of SCID miceMurine cytomegalovirus with a deletion of genes spanning HindIII-J and -I displays altered cell and tissue tropismLate expression of a beta chemokine homolog by murine cytomegalovirus.Latency versus persistence or intermittent recurrences: evidence for a latent state of murine cytomegalovirus in the lungsBiologic consequences of Stat1-independent IFN signaling.Novel cell type-specific antiviral mechanism of interferon gamma action in macrophagesMurine cytomegalovirus inhibits interferon gamma-induced antigen presentation to CD4 T cells by macrophages via regulation of expression of major histocompatibility complex class II-associated genes.Interferon gamma regulates acute and latent murine cytomegalovirus infection and chronic disease of the great vessels.Systematic mutagenesis of the murine gammaherpesvirus 68 M2 protein identifies domains important for chronic infection.
P2860
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P2860
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@ast
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@en
type
label
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@ast
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@en
prefLabel
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@ast
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney.
@en
P2860
P1433
P1476
Latency, without persistence, of murine cytomegalovirus in the spleen and kidney
@en
P2093
H W Virgin
J L Pollock
P2860
P304
P407
P577
1995-03-01T00:00:00Z