Induction of apoptosis by the severe acute respiratory syndrome coronavirus 7a protein is dependent on its interaction with the Bcl-XL protein.
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The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesisViral control of mitochondrial apoptosisThe open reading frame 3a protein of severe acute respiratory syndrome-associated coronavirus promotes membrane rearrangement and cell death.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.The N-terminal region of severe acute respiratory syndrome coronavirus protein 6 induces membrane rearrangement and enhances virus replication.Severe Acute Respiratory Syndrome Coronavirus ORF7a Inhibits Bone Marrow Stromal Antigen 2 Virion Tethering through a Novel Mechanism of Glycosylation Interference.The Non-structural Protein of Crimean-Congo Hemorrhagic Fever Virus Disrupts the Mitochondrial Membrane Potential and Induces ApoptosisRegulation of cell death during infection by the severe acute respiratory syndrome coronavirus and other coronaviruses.Anticancer compound ABT-263 accelerates apoptosis in virus-infected cells and imbalances cytokine production and lowers survival rates of infected miceSevere acute respiratory syndrome coronavirus protein 6 is required for optimal replication.Severe acute respiratory syndrome coronavirus triggers apoptosis via protein kinase R but is resistant to its antiviral activityReverse genetic characterization of the natural genomic deletion in SARS-Coronavirus strain Frankfurt-1 open reading frame 7b reveals an attenuating function of the 7b protein in-vitro and in-vivo.SARS coronavirus protein 7a interacts with human Ap4A-hydrolase.MAVS-mediated apoptosis and its inhibition by viral proteins.Severe acute respiratory syndrome coronavirus gene 7 products contribute to virus-induced apoptosis.Infectious Bronchitis Virus Infection Induces Apoptosis during Replication in Chicken Macrophage HD11 Cells.Human Coronaviruses: A Review of Virus-Host Interactions.Highly pathogenic avian influenza A virus H5N1 non-structural protein 1 is associated with apoptotic activation of the intrinsic mitochondrial pathwayInvolvement of GRIM-19 in apoptosis induced in H5N1 virus-infected human macrophages.
P2860
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P2860
Induction of apoptosis by the severe acute respiratory syndrome coronavirus 7a protein is dependent on its interaction with the Bcl-XL protein.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@ast
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@en
type
label
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@ast
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@en
prefLabel
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@ast
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@en
P2093
P2860
P50
P921
P356
P1433
P1476
Induction of apoptosis by the ...... ction with the Bcl-XL protein.
@en
P2093
Chris Birch
Marvin J-R Lee
Mike Catton
Nai Yang Fu
Puay-Yoke Tham
Timothy H P Tan
Vithiagaran Gunalan
Ying-Xim Tan
P2860
P304
P356
10.1128/JVI.00090-07
P407
P577
2007-04-11T00:00:00Z