H3K9 Trimethylation Silences Fas Expression To Confer Colon Carcinoma Immune Escape and 5-Fluorouracil Chemoresistance.
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Discovery of Anticancer Agents of Diverse Natural OriginThe Janus Face of Death Receptor Signaling during Tumor ImmunoeditingHistone lysine methyltransferases as anti-cancer targets for drug discoveryCeramide mediates FasL-induced caspase 8 activation in colon carcinoma cells to enhance FasL-induced cytotoxicity by tumor-specific cytotoxic T lymphocytesEnhanced dereplication of fungal cultures via use of mass defect filtering.New frontiers in the treatment of colorectal cancer: Autophagy and the unfolded protein response as promising targetsNF-κB functions as a molecular link between tumor cells and Th1/Tc1 T cells in the tumor microenvironment to exert radiation-mediated tumor suppression.Verticillin A Inhibits Leiomyosarcoma and Malignant Peripheral Nerve Sheath Tumor Growth via Induction of Apoptosis.Ceramide activates lysosomal cathepsin B and cathepsin D to attenuate autophagy and induces ER stress to suppress myeloid-derived suppressor cells.Plasma pharmacokinetics and bioavailability of verticillin A following different routes of administration in mice using liquid chromatography tandem mass spectrometry.Nanoparticle drug-delivery systems for peritoneal cancers: a case study of the design, characterization and development of the expansile nanoparticle.Anticancer Natural Compounds as Epigenetic Modulators of Gene ExpressionPeritoneal carcinomatosis of colorectal cancer is characterized by structural and functional reorganization of the tumor microenvironment inducing senescence and proliferation arrest in cancer cells.CRHR2/Ucn2 signaling is a novel regulator of miR-7/YY1/Fas circuitry contributing to reversal of colorectal cancer cell resistance to Fas-mediated apoptosis.Contrasting roles of H3K4me3 and H3K9me3 in regulation of apoptosis and gemcitabine resistance in human pancreatic cancer cells.Pharmacoepigenetics and pharmacoepigenomics of gastrointestinal cancers.Distinct histone modifications denote early stress-induced drug tolerance in cancer.SETD1B Activates iNOS Expression in Myeloid-Derived Suppressor Cells.microRNA-577 suppresses tumor growth and enhances chemosensitivity in colorectal cancer.p53 and metabolism: from mechanism to therapeutics.
P2860
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P2860
H3K9 Trimethylation Silences Fas Expression To Confer Colon Carcinoma Immune Escape and 5-Fluorouracil Chemoresistance.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@ast
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@en
type
label
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@ast
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@en
prefLabel
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@ast
H3K9 Trimethylation Silences F ...... -Fluorouracil Chemoresistance.
@en
P2093
P2860
P356
P1476
H3K9 Trimethylation Silences F ...... 5-Fluorouracil Chemoresistance
@en
P2093
Amy V Paschall
Asha Nayak-Kapoor
Cedric Pearce
Chunwan Lu
Dafeng Yang
Feiyan Liu
Jeong-Hyeon Choi
Wendy B Bollag
P2860
P304
P356
10.4049/JIMMUNOL.1402243
P407
P577
2015-07-01T00:00:00Z