Inflammasome-regulated cytokines are critical mediators of acute lung injury - Test2 - elhamkhani - TriplyDB

Inflammasome-regulated cytokines are critical mediators of acute lung injury

Inflammasome-regulated cytokines are critical mediators of acute lung injury

http://www.wikidata.org/entity/Q36026253

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A Cellular GWAS Approach to Define Human Variation in Cellular Pathways Important to Inflammation A Pathophysiologic Approach to Biomarkers in Acute Respiratory Distress Syndrome Animal Models, Learning Lessons to Prevent and Treat Neonatal Chronic Lung Disease Novel approaches to minimize ventilator-induced lung injury Microvascular reactivity is altered early in patients with acute respiratory distress syndrome.A comprehensive time-course-based multicohort analysis of sepsis and sterile inflammation reveals a robust diagnostic gene set Inflection points in sepsis biology: from local defense to systemic organ injury Carbon monoxide in the treatment of sepsis The acute respiratory distress syndrome: from mechanism to translation IL-1R signalling is critical for regulation of multi-walled carbon nanotubes-induced acute lung inflammation in C57Bl/6 mice Effect of MWCNT size, carboxylation, and purification on in vitro and in vivo toxicity, inflammation and lung pathology Regulation and Function of the Nucleotide Binding Domain Leucine-Rich Repeat-Containing Receptor, Pyrin Domain-Containing-3 Inflammasome in Lung Disease The Roles of Mitochondrial Damage-Associated Molecular Patterns in Diseases Circulating mitochondrial DNA in patients in the ICU as a marker of mortality: derivation and validation Severity of disease estimation and risk-adjustment for comparison of outcomes in mechanically ventilated patients using electronic routine care data.Inhibition of endotoxin-induced airway epithelial cell injury by a novel family of pyrrol derivates.Benchmarking Sepsis Gene Expression Diagnostics Using Public Data.Mitochondria in lung biology and pathology: more than just a powerhouse.AIM2 Inflammasome Is Critical for Influenza-Induced Lung Injury and Mortality Zinc deficiency primes the lung for ventilator-induced injury Pivotal role of the 5-lipoxygenase pathway in lung injury after experimental sepsis Critical role for the NLRP3 inflammasome during acute lung injury Role of P2X7R in the development and progression of pulmonary hypertension Integrative "omic" analysis of experimental bacteremia identifies a metabolic signature that distinguishes human sepsis from systemic inflammatory response syndromes Pre-treatment with allopurinol or uricase attenuates barrier dysfunction but not inflammation during murine ventilator-induced lung injury.Caspase-1 Activation Protects Lung Endothelial Barrier Function during Infection-Induced Stress.Bronchoscopy-derived correlates of lung injury following inhalational injuries: a prospective observational study.High levels of S100A8/A9 proteins aggravate ventilator-induced lung injury via TLR4 signaling.The plasma mitochondrial DNA is an independent predictor for post-traumatic systemic inflammatory response syndrome.Inflammasome Activity in Non-Microbial Lung Inflammation.UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis Oxidants in Acute and Chronic Lung Disease Vimentin regulates activation of the NLRP3 inflammasome.The Effects of Portulaca oleracea on Hypoxia-Induced Pulmonary Edema in Mice Membrane translocation of IL-33 receptor in ventilator induced lung injury Role of cytokines as a double-edged sword in sepsis Analysis of mechanical ventilation and lipopolysaccharide‑induced acute lung injury using DNA microarray analysis.Sirt1 restrains lung inflammasome activation in a murine model of sepsis.Carbon monoxide negatively regulates NLRP3 inflammasome activation in macrophages.Increased expression of neutrophil-related genes in patients with early sepsis-induced ARDS.

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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RCCM.201201-0003OC

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American Journal of Respiratory and Critical Care Medicine

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Inflammasome-regulated cytokines are critical mediators of acute lung injury

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Angela Rogers

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Anthony F Massaro

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Augustine M K Choi

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Chang Hyeok An

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David C Christiani

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Gary M Hunninghake

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Jeffrey A Haspel

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Judie Howrylak

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Kiichi Nakahira

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Laura Fredenburgh

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P2860

The inflammasomes

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

Mitogen-activated protein kinases regulate susceptibility to ventilator-induced lung injury

Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. The Acute Respiratory Distress Syndrome Network

A role for mitochondria in NLRP3 inflammasome activation

Gout-associated uric acid crystals activate the NALP3 inflammasome

Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

Cryopyrin activates the inflammasome in response to toxins and ATP

Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome

The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses

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1225-1234

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10.1164/RCCM.201201-0003OC

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Jason D Christie

Lorraine B. Ware

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223990116

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