Reduction in parvalbumin expression not loss of the parvalbumin-expressing GABA interneuron subpopulation in genetic parvalbumin and shank mouse models of autism
about
Maternal Malnutrition in the Etiopathogenesis of Psychiatric Diseases: Role of Polyunsaturated Fatty AcidsSHANK proteins: roles at the synapse and in autism spectrum disorder.Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia.Prenatal Valproate Exposure Differentially Affects Parvalbumin-Expressing Neurons and Related Circuits in the Cortex and Striatum of Mice.Decreased parvalbumin mRNA levels in cerebellar Purkinje cells in autism.Targeted Interneuron Depletion in the Dorsal Striatum Produces Autism-like Behavioral Abnormalities in Male but Not Female Mice.Neurobiological bases of autism-epilepsy comorbidity: a focus on excitation/inhibition imbalance.The Role of Interneurons in Autism and Tourette Syndrome.Basal ganglia and autism - a translational perspective.Developmental Disruption of GABAAR-Meditated Inhibition in Cntnap2 KO Mice.Parvalbumin and autism: different causes, same effect?Hippocampal GABAergic Inhibitory Interneurons.Inhibitory control of the excitatory/inhibitory balance in psychiatric disorders.Aberrant cognitive phenotypes and altered hippocampal BDNF expression related to epigenetic modifications in mice lacking the post-synaptic scaffolding protein SHANK1: Implications for autism spectrum disorder.Parvalbumin-expressing ependymal cells in rostral lateral ventricle wall adhesions contribute to aging-related ventricle stenosis in mice.Parvalbumin neurons as a hub in autism spectrum disorders.17-β estradiol increases parvalbumin levels in Pvalb heterozygous mice and attenuates behavioral phenotypes with relevance to autism core symptoms.Crossmodal Connections of Primary Sensory Cortices Largely Vanish During Normal Aging.INTERNEURONOPATHIES AND THEIR ROLE IN EARLY LIFE EPILEPSIES AND NEURODEVELOPMENTAL DISORDERS.Common Ribs of Inhibitory Synaptic Dysfunction in the Umbrella of Neurodevelopmental Disorders.Hyperactivity and Hypermotivation Associated With Increased Striatal mGluR1 Signaling in a Shank2 Rat Model of Autism.Shank2 Deletion in Parvalbumin Neurons Leads to Moderate Hyperactivity, Enhanced Self-Grooming and Suppressed Seizure Susceptibility in Mice.GABA Neuronal Deletion of Exons 14-16 in Mice Suppresses Striatal Excitatory Synaptic Input and Induces Social and Locomotor AbnormalitiesSocial Stimulus Causes Aberrant Activation of the Medial Prefrontal Cortex in a Mouse Model With Autism-Like BehaviorsThe Neuroprotective Effects of 17β-Estradiol Pretreatment in a Model of Neonatal Hippocampal Injury Induced by TrimethyltinDecreased Number and Expression of nNOS-Positive Interneurons in Basolateral Amygdala in Two Mouse Models of AutismDysregulation of Parvalbumin Expression in the Mouse Model of Autism Spectrum DisorderMice With Decreased Number of Interneurons Exhibit Aberrant Spontaneous and Oscillatory Activity in the Cortex
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P2860
Reduction in parvalbumin expression not loss of the parvalbumin-expressing GABA interneuron subpopulation in genetic parvalbumin and shank mouse models of autism
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Reduction in parvalbumin expre ...... d shank mouse models of autism
@ast
Reduction in parvalbumin expre ...... d shank mouse models of autism
@en
type
label
Reduction in parvalbumin expre ...... d shank mouse models of autism
@ast
Reduction in parvalbumin expre ...... d shank mouse models of autism
@en
prefLabel
Reduction in parvalbumin expre ...... d shank mouse models of autism
@ast
Reduction in parvalbumin expre ...... d shank mouse models of autism
@en
P2860
P50
P1433
P1476
Reduction in parvalbumin expre ...... d shank mouse models of autism
@en
P2093
Karl Jakob Vörckel
P2860
P2888
P356
10.1186/S13041-016-0192-8
P577
2016-01-27T00:00:00Z
P5875
P6179
1053199562