Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
about
Baicalin suppresses NLRP3 inflammasome and nuclear factor-kappa B (NF-κB) signaling during Haemophilus parasuis infectionMacrophages in gastrointestinal homeostasis and inflammation.Inflammasomes and intestinal inflammation.Interleukin 1β Mediates Intestinal Inflammation in Mice and Patients With Interleukin 10 Receptor Deficiency.Remodelling of the gut microbiota by hyperactive NLRP3 induces regulatory T cells to maintain homeostasis.The Stimulation of Macrophages with TLR Ligands Supports Increased IL-19 Expression in Inflammatory Bowel Disease Patients and in Colitis Models.The RNA-binding protein Tristetraprolin (TTP) is a critical negative regulator of the NLRP3 inflammasome.
P2860
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P2860
Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
@en
type
label
Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
@en
prefLabel
Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
@en
P2093
P2860
P356
P1433
P1476
Posttranscriptional control of NLRP3 inflammasome activation in colonic macrophages.
@en
P2093
P2860
P2888
P304
P356
10.1038/MI.2015.109
P577
2015-12-02T00:00:00Z