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The SMC-5/6 Complex and the HIM-6 (BLM) Helicase Synergistically Promote Meiotic Recombination Intermediate Processing and Chromosome Maturation during Caenorhabditis elegans MeiosisStructure of the RecQ C-terminal domain of human Bloom syndrome proteinCharacterization of the Caenorhabditis elegans HIM-6/BLM helicase: unwinding recombination intermediatesConstruction of DNA hemicatenanes from two small circular DNA moleculesRegulation of BLM Nucleolar LocalizationThe dissolution of double Holliday junctions.Protein degradation pathways regulate the functions of helicases in the DNA damage response and maintenance of genomic stabilityHuman RecQ helicases in DNA repair, recombination, and replication.Accumulation and Phosphorylation of RecQ-Mediated Genome Instability Protein 1 (RMI1) at Serine 284 and Serine 292 during Mitosis.hSSB1 associates with and promotes stability of the BLM helicaseTargeting SOD1 induces synthetic lethal killing in BLM- and CHEK2-deficient colorectal cancer cells.A High-Throughput Screening Strategy to Identify Protein-Protein Interaction Inhibitors That Block the Fanconi Anemia DNA Repair Pathway.RecD2 helicase limits replication fork stress in Bacillus subtilis.Quality control of homologous recombination.Molecular mechanism of double Holliday junction dissolution.Structural studies of DNA end detection and resection in homologous recombination.Holliday junction resolvases.Stress and DNA repair biology of the Fanconi anemia pathwayStructural mechanisms of human RecQ helicases WRN and BLM.Effects of Adenovirus Type 5 E1A Isoforms on Viral Replication in Arrested Human CellsSmc5/6 complex regulates Sgs1 recombination functions.BLM promotes the activation of Fanconi Anemia signaling pathway.Bloom syndrome radials are predominantly non-homologous and are suppressed by phosphorylated BLMStructure and Function of a Novel ATPase that Interacts with Holliday Junction Resolvase Hjc and Promotes Branch Migration.Defining the roles of the N-terminal region and the helicase activity of RECQ4A in DNA repair and homologous recombination in Arabidopsis.Mouse embryonic stem cells have increased capacity for replication fork restart driven by the specific Filia-Floped protein complex.BLM helicase regulates DNA repair by counteracting RAD51 loading at DNA double-strand break sites.A tri-serine cluster within the topoisomerase IIα-interaction domain of the BLM helicase is required for regulating chromosome breakage in human cells.
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description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 31 March 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The BLM dissolvasome in DNA replication and repair.
@en
The BLM dissolvasome in DNA replication and repair.
@nl
type
label
The BLM dissolvasome in DNA replication and repair.
@en
The BLM dissolvasome in DNA replication and repair.
@nl
prefLabel
The BLM dissolvasome in DNA replication and repair.
@en
The BLM dissolvasome in DNA replication and repair.
@nl
P2860
P1476
The BLM dissolvasome in DNA replication and repair
@en
P2093
Kelly A Manthei
P2860
P2888
P304
P356
10.1007/S00018-013-1325-1
P577
2013-03-31T00:00:00Z