A KCNQ channel opener for experimental neonatal seizures and status epilepticus - Test2 - elhamkhani - TriplyDB

A KCNQ channel opener for experimental neonatal seizures and status epilepticus

A KCNQ channel opener for experimental neonatal seizures and status epilepticus

http://www.wikidata.org/entity/Q37151699

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Searching for new targets for treatment of pediatric epilepsy M-current preservation contributes to anticonvulsant effects of valproic acid.Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potential Retigabine, a Kv7.2/Kv7.3-Channel Opener, Attenuates Drug-Induced Seizures in Knock-In Mice Harboring Kcnq2 Mutations Enhanced AMPA receptor-mediated neurotransmission on CA1 pyramidal neurons during status epilepticus.Phosphatase inhibition prevents the activity-dependent trafficking of GABAA receptors during status epilepticus in the young animal.Impact of transient acute hypoxia on the developing mouse EEG K(+) channelepsy: progress in the neurobiology of potassium channels and epilepsy.Development of later life spontaneous seizures in a rodent model of hypoxia-induced neonatal seizures Bumetanide enhances phenobarbital efficacy in a rat model of hypoxic neonatal seizures.Progressive NKCC1-dependent neuronal chloride accumulation during neonatal seizures.Neuronal potassium channel openers in the management of epilepsy: role and potential of retigabine.The Effects of the KCNQ Openers Retigabine and Flupirtine on Myotonia in Mammalian Skeletal Muscle Induced by a Chloride Channel Blocker.Neonatal seizures: controversies and challenges in translating new therapies from the lab to the isolette.Flupirtine effectively prevents development of acute neonatal seizures in an animal model of global hypoxia.Ischemic injury suppresses hypoxia-induced electrographic seizures and the background EEG in a rat model of perinatal hypoxic-ischemic encephalopathy.Recording EEG in immature rats with a novel miniature telemetry system.Genotype-phenotype correlations in neonatal epilepsies caused by mutations in the voltage sensor of K(v)7.2 potassium channel subunits.Potassium Channels in Epilepsy.Expression and function of KCNQ channels in larval zebrafish.Flupirtine and diazepam combination terminates established status epilepticus: results in three rodent models.Neonatal Seizures

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A KCNQ channel opener for experimental neonatal seizures and status epilepticus

http://www.wikidata.org/entity/Q37151699

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

A KCNQ channel opener for experimental neonatal seizures and status epilepticus

@en

A KCNQ channel opener for experimental neonatal seizures and status epilepticus.

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type

label

A KCNQ channel opener for experimental neonatal seizures and status epilepticus

@en

A KCNQ channel opener for experimental neonatal seizures and status epilepticus.

@nl

prefLabel

A KCNQ channel opener for experimental neonatal seizures and status epilepticus

@en

A KCNQ channel opener for experimental neonatal seizures and status epilepticus.

@nl

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Annals of Neurology

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A KCNQ channel opener for experimental neonatal seizures and status epilepticus

@en

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Amy R Brooks-Kayal

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David A Lapides

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Edward C Cooper

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Jeffery G Keating

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Yogendrasinh H Raol

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P2860

A common ankyrin-G-based mechanism retains KCNQ and NaV channels at electrically active domains of the axon

Antiepileptic drugs and apoptotic neurodegeneration in the developing brain

Genetic heterogeneity in benign familial neonatal convulsions: identification of a new locus on chromosome 8q

Colocalization and coassembly of two human brain M-type potassium channel subunits that are mutated in epilepsy

KCNQ2 and KCNQ3 potassium channel genes in benign familial neonatal convulsions: expansion of the functional and mutation spectrum

Andreas Rett and benign familial neonatal convulsions revisited

NKCC1 transporter facilitates seizures in the developing brain

Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy

Developmental seizures induced by common early-life insults: short- and long-term effects on seizure susceptibility

Flupirtine blocks apoptosis in batten patient lymphoblasts and in human postmitotic CLN3- and CLN2-deficient neurons.

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326-336

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scholarly article

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10.1002/ANA.21593

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3

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65

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2009-03-01T00:00:00Z

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24246331

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19334075

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status epilepticus

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2666110

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