A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response. - Test2 - elhamkhani - TriplyDB

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

http://www.wikidata.org/entity/Q37285664

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Shiga Toxin Therapeutics: Beyond Neutralization.Treatments for Pulmonary Ricin Intoxication: Current Aspects and Future Prospects.

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A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

http://www.wikidata.org/entity/Q37285664

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 September 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@en

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@nl

type

label

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@en

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@nl

prefLabel

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@en

A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@nl

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A Novel Zak Knockout Mouse with a Defective Ribotoxic Stress Response.

@en

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Bruce E Magun

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Cheleste M Thorpe

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Dakshina M Jandhyala

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John M Leong

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P2860

MRK, a mixed lineage kinase-related molecule that plays a role in gamma-radiation-induced cell cycle arrest

The stress kinase MRK contributes to regulation of DNA damage checkpoints through a p38gamma-independent pathway

Investigation of a ricin-containing envelope at a postal facility - South Carolina, 2003

Identification and characterization of a novel MAP kinase kinase kinase, MLTK

Tissue distribution and functional expression of a cDNA encoding a novel mixed lineage kinase

Complete inhibition of anisomycin and UV radiation but not cytokine induced JNK and p38 activation by an aryl-substituted dihydropyrrolopyrazole quinoline and mixed lineage kinase 7 small interfering RNA

Shiga toxins stimulate secretion of interleukin-8 from intestinal epithelial cells.

Administration of ricin induces a severe inflammatory response via nonredundant stimulation of ERK, JNK, and P38 MAPK and provides a mouse model of hemolytic uremic syndrome.

Long non-coding RNA URHC regulates cell proliferation and apoptosis via ZAK through the ERK/MAPK signaling pathway in hepatocellular carcinoma.

Shiga toxins induce, superinduce, and stabilize a variety of C-X-C chemokine mRNAs in intestinal epithelial cells, resulting in increased chemokine expression

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scholarly article

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10.3390/TOXINS8090259

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9

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8

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Nicholas J. Mantis

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2016-09-02T00:00:00Z

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27598200

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