Loss of AF6/afadin, a marker of poor outcome in breast cancer, induces cell migration, invasiveness and tumor growth.
about
Alterations of the apical junctional complex and actin cytoskeleton and their role in colorectal cancer progressionAfadin is localized at cell-cell contact sites in mesangial cells and regulates migratory polarity.Genome-wide characterization of pancreatic adenocarcinoma patients using next generation sequencing.The Expression of the Zonula Adhaerens Protein PLEKHA7 Is Strongly Decreased in High Grade Ductal and Lobular Breast Carcinomas.Loss of AF-6/afadin induces cell invasion, suppresses the formation of glandular structures and might be a predictive marker of resistance to chemotherapy in endometrial cancerA short splicing isoform of afadin suppresses the cortical axon branching in a dominant-negative manner.Rap1 and Canoe/afadin are essential for establishment of apical-basal polarity in the Drosophila embryo.A role for PVRL4-driven cell-cell interactions in tumorigenesis.The adherens junction protein afadin is an AKT substrate that regulates breast cancer cell migration.Actin-tethered junctional complexes in angiogenesis and lymphangiogenesis in association with vascular endothelial growth factor.Loss of polarity protein AF6 promotes pancreatic cancer metastasis by inducing Snail expression.R-Ras controls axon branching through afadin in cortical neurons.Genome-Wide miRNA Analysis Identifies miR-188-3p as a Novel Prognostic Marker and Molecular Factor Involved in Colorectal Carcinogenesis.A network-based analysis of colon cancer splicing changes reveals a tumorigenesis-favoring regulatory pathway emanating from ELK1Decreased expression of the long non-coding RNA MLLT4 antisense RNA 1 is a potential biomarker and an indicator of a poor prognosis for gastric cancer.Elementary: breast cancer culprits leave their signatures on the double helix.Expression and clinical significance of Nectin-4 in hepatocellular carcinomaSynergism between canoe and scribble mutations causes tumor-like overgrowth via Ras activation in neural stem cells and epithelia.s-Afadin binds more preferentially to the cell adhesion molecules nectins than l-afadin.PI3K/AKT/Afadin signaling pathway contributes to pathological vascularization in glioblastomas.Phosphatidylserine enhances IKBKAP transcription by activating the MAPK/ERK signaling pathway.
P2860
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P2860
Loss of AF6/afadin, a marker of poor outcome in breast cancer, induces cell migration, invasiveness and tumor growth.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@en
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@nl
type
label
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@en
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@nl
prefLabel
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@en
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth.
@nl
P2093
P2860
P356
P1433
P1476
Loss of AF6/afadin, a marker o ...... invasiveness and tumor growth
@en
P2093
A Restouin
D Isnardon
E Josselin
G Fournier
J Adélaïde
M Chaffanet
R Castellano
P2860
P2888
P304
P356
10.1038/ONC.2011.106
P407
P577
2011-04-11T00:00:00Z