RAD18 promotes DNA double-strand break repair during G1 phase through chromatin retention of 53BP1.
about
Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modificationsPUB-NChIP--"in vivo biotinylation" approach to study chromatin in proximity to a protein of interestBMI1-mediated histone ubiquitylation promotes DNA double-strand break repairHuman RAD18 interacts with ubiquitylated chromatin components and facilitates RAD9 recruitment to DNA double strand breaksSystematic E2 screening reveals a UBE2D-RNF138-CtIP axis promoting DNA repairMsl2 is a novel component of the vertebrate DNA damage response53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaksRNF4, a SUMO-targeted ubiquitin E3 ligase, promotes DNA double-strand break repair.Comparative analysis of zinc finger proteins involved in plant disease resistanceThe E3 ubiquitin ligase RAD18 regulates ubiquitylation and chromatin loading of FANCD2 and FANCIRAD18 activates the G2/M checkpoint through DNA damage signaling to maintain genome integrity after ionizing radiation exposure.Regulation of DNA end joining, resection, and immunoglobulin class switch recombination by 53BP1.Meiotic functions of RAD18.c-Jun N-terminal kinase-mediated Rad18 phosphorylation facilitates Polη recruitment to stalled replication forksRNF168 forms a functional complex with RAD6 during the DNA damage responseRad18-dependent SUMOylation of human specialized DNA polymerase eta is required to prevent under-replicated DNA.RNF168 ubiquitylates 53BP1 and controls its response to DNA double-strand breaks.Eukaryotic DNA damage checkpoint activation in response to double-strand breaks.Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end joining.Rad18 E3 ubiquitin ligase activity mediates Fanconi anemia pathway activation and cell survival following DNA Topoisomerase 1 inhibition.Reading chromatin signatures after DNA double-strand breaks.Mechanisms of Ubiquitin-Nucleosome Recognition and Regulation of 53BP1 Chromatin Recruitment by RNF168/169 and RAD18.Recruitment of lysine demethylase 2A to DNA double strand breaks and its interaction with 53BP1 ensures genome stability.Tumor suppressor miR-145 reverses drug resistance by directly targeting DNA damage-related gene RAD18 in colorectal cancer.
P2860
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P2860
RAD18 promotes DNA double-strand break repair during G1 phase through chromatin retention of 53BP1.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
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2009年學術文章
@zh-hant
name
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@en
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@nl
type
label
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@en
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@nl
prefLabel
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@en
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@nl
P2093
P2860
P356
P1476
RAD18 promotes DNA double-stra ...... chromatin retention of 53BP1.
@en
P2093
Jinghua Sun
Kazuaki Tokunaga
Kenji Watanabe
Kuniyoshi Iwabuchi
Masaru Yamaizumi
Mitsumasa Hashimoto
Satoshi Tateishi
Takayasu Date
Tokio Tani
Yuri Tsuji
P2860
P304
P356
10.1093/NAR/GKP082
P577
2009-02-19T00:00:00Z