Tel, a frequent target of leukemic translocations, induces cellular aggregation and influences expression of extracellular matrix components.
about
The novel ETS factor TEL2 cooperates with Myc in B lymphomagenesisETV6 in hematopoiesis and leukemia predisposition.A detailed transcriptional map of the chromosome 12p12 tumour suppressor locus.Small ubiquitin-like modifier conjugation regulates nuclear export of TEL, a putative tumor suppressorIdentification of transcripts modulated by ETV6 expression.TEL/ETV6 is a signal transducer and activator of transcription 3 (Stat3)-induced repressor of Stat3 activity.The MN1-TEL fusion protein, encoded by the translocation (12;22)(p13;q11) in myeloid leukemia, is a transcription factor with transforming activity.Tel induces a G1 arrest and suppresses Ras-induced transformation.TEL, a putative tumor suppressor, induces apoptosis and represses transcription of Bcl-XL.TEL contacts multiple co-repressors and specifically associates with histone deacetylase-3.Sumoylation and Its Contribution to Cancer.
P2860
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P2860
Tel, a frequent target of leukemic translocations, induces cellular aggregation and influences expression of extracellular matrix components.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Tel, a frequent target of leuk ...... tracellular matrix components.
@en
type
label
Tel, a frequent target of leuk ...... tracellular matrix components.
@en
prefLabel
Tel, a frequent target of leuk ...... tracellular matrix components.
@en
P2093
P2860
P356
P1433
P1476
Tel, a frequent target of leuk ...... tracellular matrix components.
@en
P2093
P2860
P304
P356
10.1038/SJ.NEO.7900064
P577
1999-12-01T00:00:00Z