The apoptosome pathway to caspase activation in primary human neutrophils exhibits dramatically reduced requirements for cytochrome C
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Mathematical modeling identifies inhibitors of apoptosis as mediators of positive feedback and bistabilityProliferating cell nuclear antigen acts as a cytoplasmic platform controlling human neutrophil survivalNADPH oxidase (NOX) isoforms are inhibited by celastrol with a dual mode of action.Pivotal Advance: Pharmacological manipulation of inflammation resolution during spontaneously resolving tissue neutrophilia in the zebrafish.Caspase-dependent inactivation of proteasome function during programmed cell death in Drosophila and man.Mitochondrial membrane potential in human neutrophils is maintained by complex III activity in the absence of supercomplex organisationStaphylococcus aureus Panton-Valentine leukocidin directly targets mitochondria and induces Bax-independent apoptosis of human neutrophils.Peculiarities of cell death mechanisms in neutrophils.Molecular mechanisms underlying delayed apoptosis in neutrophils from multiple trauma patients with and without sepsisRole of neutrophils in innate immunity: a systems biology-level approachBig wheel keeps on turning: apoptosome regulation and its role in chemoresistance.Necrostatin-1 enhances the resolution of inflammation by specifically inducing neutrophil apoptosisNeutrophil apoptosis during viral infections.Bacteria induce prolonged PMN survival via a phosphatidylcholine-specific phospholipase C- and protein kinase C-dependent mechanism.Mechanisms regulating neutrophil survival and cell death.Mcl-1 is vital for neutrophil survival.Delayed apoptosis of tumor associated neutrophils in the absence of endogenous IFN-β.Arsenic trioxide (AT) is a novel human neutrophil pro-apoptotic agent: effects of catalase on AT-induced apoptosis, degradation of cytoskeletal proteins and de novo protein synthesis.Systems analysis of effector caspase activation and its control by X-linked inhibitor of apoptosis proteinGranulocyte colony-stimulating factor delays neutrophil apoptosis by inhibition of calpains upstream of caspase-3.Acute CD47 Blockade During Ischemic Myocardial Reperfusion Enhances Phagocytosis-Associated Cardiac RepairInterchain proteolysis, in the absence of a dimerization stimulus, can initiate apoptosis-associated caspase-8 activation.Mechanisms involved in methylmercuric chloride (MeHgCl)-induced suppression of human neutrophil apoptosis.Targeting survivin via PI3K but not c-akt/PKB by anticancer drugs in immature neutrophils.Granulocyte Colony-Stimulating Factor and Granulocyte-Macrophage Colony-Stimulating Factor Have Differential Effects on Neonatal and Adult Neutrophil Survival and Function
P2860
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P2860
The apoptosome pathway to caspase activation in primary human neutrophils exhibits dramatically reduced requirements for cytochrome C
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
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2003年论文
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2003年论文
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name
The apoptosome pathway to casp ...... requirements for cytochrome C
@en
The apoptosome pathway to casp ...... requirements for cytochrome C
@nl
type
label
The apoptosome pathway to casp ...... requirements for cytochrome C
@en
The apoptosome pathway to casp ...... requirements for cytochrome C
@nl
prefLabel
The apoptosome pathway to casp ...... requirements for cytochrome C
@en
The apoptosome pathway to casp ...... requirements for cytochrome C
@nl
P2860
P50
P356
P1476
The apoptosome pathway to casp ...... requirements for cytochrome C
@en
P2093
Amanda J O'Neill
R William G Watson
P2860
P304
P356
10.1084/JEM.20021862
P407
P577
2003-03-01T00:00:00Z