Bidirectional synaptic plasticity and spatial memory flexibility require Ca2+-stimulated adenylyl cyclases.
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Hippocampal Transcriptome Profile of Persistent Memory Rescue in a Mouse Model of THRA1 Mutation-Mediated Resistance to Thyroid Hormone.The sodium-activated potassium channel Slack is required for optimal cognitive flexibility in miceType VI adenylyl cyclase negatively regulates GluN2B-mediated LTD and spatial reversal learning.Activity-dependent p25 generation regulates synaptic plasticity and Aβ-induced cognitive impairmentTet1 is critical for neuronal activity-regulated gene expression and memory extinction.Elevated activation of CaMKIIα in the CPEB3-knockout hippocampus impairs a specific form of NMDAR-dependent synaptic depotentiationPKMζ differentially utilized between sexes for remote long-term spatial memory.Absence of Ca2+-stimulated adenylyl cyclases leads to reduced synaptic plasticity and impaired experience-dependent fear memoryRegulation of brain-derived neurotrophic factor expression in neurons.Modelling intracellular competition for calcium: kinetic and thermodynamic control of different molecular modes of signal decodingRobust training attenuates TBI-induced deficits in reference and working memory on the radial 8-arm maze.Increases in cAMP, MAPK activity, and CREB phosphorylation during REM sleep: implications for REM sleep and memory consolidationRegulating the ubiquitin/proteasome pathway via cAMP-signaling: neuroprotective potentialDeficits in behavioral sensitization and dopaminergic responses to methamphetamine in adenylyl cyclase 1/8-deficient mice.Depotentiation from Potentiated Synaptic Strength in a Tristable System of Coupled Phosphatase and KinaseNMDA receptor-dependent long-term potentiation comprises a family of temporally overlapping forms of synaptic plasticity that are induced by different patterns of stimulation.Voluntary running depreciates the requirement of Ca2+-stimulated cAMP signaling in synaptic potentiation and memory formation.International Union of Basic and Clinical Pharmacology. CI. Structures and Small Molecule Modulators of Mammalian Adenylyl Cyclases.Calmodulin activity regulates group I metabotropic glutamate receptor-mediated signal transduction and synaptic depression.Acute inhibition of mGluR5 disrupts behavioral flexibility.β-adrenergic signaling broadly contributes to LTP induction.Mice overexpressing type 1 adenylyl cyclase show enhanced spatial memory flexibility in the absence of intact synaptic long-term depression.Separate Ionotropic and Metabotropic Glutamate Receptor Functions in Depotentiation vs. LTP: A Distinct Role for Group1 mGluR Subtypes and NMDARs.Switching off LTP: mGlu and NMDA receptor-dependent novelty exploration-induced depotentiation in the rat hippocampus.Presynaptic kainate receptor-mediated facilitation of glutamate release involves Ca2+ -calmodulin at mossy fiber-CA3 synapses.Ca2+-Binding Protein 1 Regulates Hippocampal-dependent Memory and Synaptic Plasticity.Non-canonical Mechanisms of Presynaptic Kainate Receptors Controlling Glutamate Release.
P2860
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P2860
Bidirectional synaptic plasticity and spatial memory flexibility require Ca2+-stimulated adenylyl cyclases.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@en
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@nl
type
label
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@en
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@nl
prefLabel
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@en
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@nl
P2093
P2860
P1476
Bidirectional synaptic plastic ...... -stimulated adenylyl cyclases.
@en
P2093
Daniel R Storm
Hongbing Wang
Ming Zhang
P2860
P304
10174-10183
P356
10.1523/JNEUROSCI.0009-11.2011
P407
P577
2011-07-01T00:00:00Z