Characterization of the mechanism underlying the reversal of long term potentiation by low frequency stimulation at hippocampal CA1 synapses.
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Multiple forms of metaplasticity at a single hippocampal synapse during late postnatal developmentSTDP in a bistable synapse model based on CaMKII and associated signaling pathwaysTemporal sensitivity of protein kinase a activation in late-phase long term potentiationCofilin Activation Is Temporally Associated with the Cessation of Growth in the Developing HippocampusA single high dose of dexamethasone affects the phosphorylation state of glutamate AMPA receptors in the human limbic systemDynamic synapses as archives of synaptic history: state-dependent redistribution of synaptic efficacy in the rat hippocampal CA1.Experimental and computational aspects of signaling mechanisms of spike-timing-dependent plasticity.Specific roles of AMPA receptor subunit GluR1 (GluA1) phosphorylation sites in regulating synaptic plasticity in the CA1 region of hippocampusLong-term synaptic plasticity is impaired in rats with lesions of the ventrolateral preoptic nucleus.Hippocampal short- and long-term plasticity are not modulated by astrocyte Ca2+ signaling.MHC class I immune proteins are critical for hippocampus-dependent memory and gate NMDAR-dependent hippocampal long-term depression.Reversal of plasticity-like effects in the human motor cortexElevated activation of CaMKIIα in the CPEB3-knockout hippocampus impairs a specific form of NMDAR-dependent synaptic depotentiationDifferent mechanisms of fear extinction dependent on length of time since fear acquisition.Integrin-driven actin polymerization consolidates long-term potentiation"Silent" metaplasticity of the late phase of long-term potentiation requires protein phosphatases.Trisomy of the G protein-coupled K+ channel gene, Kcnj6, affects reward mechanisms, cognitive functions, and synaptic plasticity in mice.The molecular pharmacology and cell biology of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptorsMolecular mechanisms involved in depotentiation and their relevance to schizophrenia.Regulation of GluA1 AMPA receptor through PKC phosphorylation induced by free fatty acid derivative HUHS2002.Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent MannerActivation of α7 nicotinic acetylcholine receptors protects potentiated synapses from depotentiation during theta pattern stimulation in the hippocampal CA1 region of ratsA role of p38 mitogen-activated protein kinase in adenosine A₁ receptor-mediated synaptic depotentiation in area CA1 of the rat hippocampus.G protein-activated inwardly rectifying potassium channels mediate depotentiation of long-term potentiation.Reversal of long-term potentiation-like plasticity processes after motor learning disrupts skill retention.Immediate extinction causes a less durable loss of performance than delayed extinction following either fear or appetitive conditioningAdenosine A(1) receptor: Functional receptor-receptor interactions in the brain.A structural mechanism for maintaining the 'on-state' of the CaMKII memory switch in the post-synaptic density.Roles of phosphotase 2A in nociceptive signal processing.Synaptic plasticity: a unifying model to address some persisting questions.GluA2-dependent AMPA receptor endocytosis and the decay of early and late long-term potentiation: possible mechanisms for forgetting of short- and long-term memories.In vitro reconstitution of a CaMKII memory switch by an NMDA receptor-derived peptide.EPO induces changes in synaptic transmission and plasticity in the dentate gyrus of rats.Silence analysis of AMPA receptor mutated at the CaM-kinase II phosphorylation site.Separate Ionotropic and Metabotropic Glutamate Receptor Functions in Depotentiation vs. LTP: A Distinct Role for Group1 mGluR Subtypes and NMDARs.Direct cortical inputs erase long-term potentiation at Schaffer collateral synapses.Coupled phosphatase and kinase switches produce the tristability required for long-term potentiation and long-term depression.Coexistence of muscarinic long-term depression with electrically induced long-term potentiation and depression at CA3-CA1 synapses.Regulation of glutamate receptor trafficking by leptin.Leptin reverses long-term potentiation at hippocampal CA1 synapses.
P2860
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P2860
Characterization of the mechanism underlying the reversal of long term potentiation by low frequency stimulation at hippocampal CA1 synapses.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@en
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@nl
type
label
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@en
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@nl
prefLabel
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@en
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@nl
P2093
P2860
P356
P1476
Characterization of the mechan ...... n at hippocampal CA1 synapses.
@en
P2093
P2860
P304
48108-48117
P356
10.1074/JBC.M106388200
P407
P577
2001-10-25T00:00:00Z