A key role of the hSNF5/INI1 tumour suppressor in the control of the G1-S transition of the cell cycle.
about
Residual complexes containing SMARCA2 (BRM) underlie the oncogenic drive of SMARCA4 (BRG1) mutationMutation of the INI1 gene in composite rhabdoid tumor of the endometriumApoptotic response of malignant rhabdoid tumor cellsGene of the month: SMARCB1Reverse genetic analysis of the yeast RSC chromatin remodeler reveals a role for RSC3 and SNF5 homolog 1 in ploidy maintenanceThe SWI/SNF chromatin-remodeling complex subunit SNF5 is essential for hepatocyte differentiationGeneration of a mouse model of atypical teratoid/rhabdoid tumor of the central nervous system through combined deletion of Snf5 and p53.Imprinted CDKN1C is a tumor suppressor in rhabdoid tumor and activated by restoration of SMARCB1 and histone deacetylase inhibitorsOncogenic roles of SMARCB1/INI1 and its deficient tumorsReexpression of hSNF5 in malignant rhabdoid tumor cell lines causes cell cycle arrest through a p21(CIP1/WAF1)-dependent mechanismPotent inhibition of rhabdoid tumor cells by combination of flavopiridol and 4OH-tamoxifen.The BRG1- and hBRM-associated factor BAF57 induces apoptosis by stimulating expression of the cylindromatosis tumor suppressor gene.Morphologic and molecular characterization of ATRT xenografts adapted for orthotopic therapeutic testing.Renal medullary carcinoma: case report of an aggressive malignancy with near-complete response to dose-dense methotrexate, vinblastine, Doxorubicin, and Cisplatin chemotherapyInactivation of the Snf5 tumor suppressor stimulates cell cycle progression and cooperates with p53 loss in oncogenic transformation.The SWI/SNF subunit/tumor suppressor BAF47/INI1 is essential in cell cycle arrest upon skeletal muscle terminal differentiation.Mechanisms by which SMARCB1 loss drives rhabdoid tumor growth.SNR1 (INI1/SNF5) mediates important cell growth functions of the Drosophila Brahma (SWI/SNF) chromatin remodeling complex.Chromatin remodeling and cancer, Part II: ATP-dependent chromatin remodeling.The histone deacetylase inhibitor SAHA acts in synergism with fenretinide and doxorubicin to control growth of rhabdoid tumor cells.Fibroblast growth factor receptors as novel therapeutic targets in SNF5-deleted malignant rhabdoid tumorsp16INK4A and p14ARF tumor suppressor pathways are deregulated in malignant rhabdoid tumors.Genetic interaction mapping reveals a role for the SWI/SNF nucleosome remodeler in spliceosome activation in fission yeastRhabdoid Variant of Myoepithelial Carcinoma, with EWSR1 Rearrangement: Expanding the Spectrum of EWSR1-Rearranged Myoepithelial Tumors.Nuclear transport and cancer: from mechanism to intervention.Successful salvage and long-term survival after recurrent malignant rhabdoid tumorAtypical teratoid rhabdoid tumor: current therapy and future directions.Role of proteomics in translational research in cervical cancer.Surgery and actinomycin improve survival in malignant rhabdoid tumorWhole Exome- and mRNA-Sequencing of an AT/RT Case Reveals Few Somatic Mutations and Several Deregulated Signalling Pathways in the Context of SMARCB1 Deficiency.Establishment and characterization of MRT cell lines from genetically engineered mouse models and the influence of genetic background on their development.Polybromo-1: the chromatin targeting subunit of the PBAF complex.Oncogenesis caused by loss of the SNF5 tumor suppressor is dependent on activity of BRG1, the ATPase of the SWI/SNF chromatin remodeling complex.The role of INI1/hSNF5 in gene regulation and cancer.The role of HPV E6 and E7 oncoproteins in HPV-associated cervical carcinogenesisThe role of maintenance proteins in the preservation of epithelial cell identity during mammary gland remodeling and breast cancer initiation.Throwing the cancer switch: reciprocal roles of polycomb and trithorax proteinsAtypical teratoid/rhabdoid tumor: short clinical description and insight into possible mechanism of the disease.Proteomics strategies to analyze HPV-transformed cells: relevance to cervical cancer.Molecular biology of pediatric brain tumors and impact on novel therapies.
P2860
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P2860
A key role of the hSNF5/INI1 tumour suppressor in the control of the G1-S transition of the cell cycle.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@en
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@nl
type
label
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@en
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@nl
prefLabel
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@en
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@nl
P2093
P2860
P356
P1433
P1476
A key role of the hSNF5/INI1 t ...... transition of the cell cycle.
@en
P2093
Danny Rouillard
Isabella Versteege
Souhila Medjkane
P2860
P2888
P304
P356
10.1038/SJ.ONC.1205841
P407
P577
2002-09-01T00:00:00Z