Epithelial sodium channel silencing as a strategy to correct the airway surface fluid deficit in cystic fibrosis.
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Emphysema Is Common in Lungs of Cystic Fibrosis Lung Transplantation Patients: A Histopathological and Computed Tomography Study.Direct interaction of a CFTR potentiator and a CFTR corrector with phospholipid bilayers.High-throughput screening identifies FAU protein as a regulator of mutant cystic fibrosis transmembrane conductance regulator channel.The Epithelial Sodium Channel (αENaC) Is a Downstream Therapeutic Target of ASCL1 in Pulmonary Neuroendocrine Tumors.Effective silencing of ENaC by siRNA delivered with epithelial-targeted nanocomplexes in human cystic fibrosis cells and in mouse lung.Anion-Transport Mechanism of a Triazole-Bearing Derivative of Prodigiosine: A Candidate for Cystic Fibrosis Therapy
P2860
Epithelial sodium channel silencing as a strategy to correct the airway surface fluid deficit in cystic fibrosis.
description
2013 nî lūn-bûn
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2013年の論文
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name
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@en
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@nl
type
label
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@en
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@nl
prefLabel
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@en
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@nl
P2093
P2860
P356
P1476
Epithelial sodium channel sile ...... id deficit in cystic fibrosis.
@en
P2093
Ambra Gianotti
Elvira Sondo
Emanuela Caci
Luis J V Galietta
Olga Zegarra-Moran
Raffaella Melani
Roberto Ravazzolo
P2860
P304
P356
10.1165/RCMB.2012-0408OC
P577
2013-09-01T00:00:00Z