ALK1 signaling in development and disease: new paradigms.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

ALK1 signaling in development and disease: new paradigms.

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ALK1 signaling in development and disease: new paradigms.

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ALK1 signaling in development and disease: new paradigms.

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ALK1 signaling in development and disease: new paradigms.

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ALK1 signaling in development and disease: new paradigms.

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ALK1 signaling in development and disease: new paradigms.

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P1476

ALK1 signaling in development and disease: new paradigms

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P2093

Andrew P Hinck

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Beth L Roman

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P2860

Endoglin is an accessory protein that interacts with the signaling receptor complex of multiple members of the transforming growth factor-beta superfamily

Heart specific expression of mouse BMP-10 a novel member of the TGF-beta superfamily

Tmem100, an ALK1 receptor signaling-dependent gene essential for arterial endothelium differentiation and vascular morphogenesis

Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

Molecular and functional analysis identifies ALK-1 as the predominant cause of pulmonary hypertension related to hereditary haemorrhagic telangiectasia

BMP-9 induces proliferation of multiple types of endothelial cells in vitro and in vivo

Identification of BMP9 and BMP10 as functional activators of the orphan activin receptor-like kinase 1 (ALK1) in endothelial cells

Endostatin: an endogenous inhibitor of angiogenesis and tumor growth

Bone morphogenetic protein (BMP) and activin type II receptors balance BMP9 signals mediated by activin receptor-like kinase-1 in human pulmonary artery endothelial cells

Endoglin, a TGF-beta binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1

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scholarly article

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10.1007/S00018-017-2636-4

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Beth L Roman

Andrew P Hinck

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2017-09-04T00:00:00Z

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