Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
about
Ion channel dysfunction in cerebellar ataxia.Precise Excision of the CAG Tract from the Huntingtin Gene by Cas9 Nickases.A Huntingtin Knockin Pig Model Recapitulates Features of Selective Neurodegeneration in Huntington's Disease.Recognition of the polycistronic nature of human genes is critical to understanding the genotype-phenotype relationship.Purinergic Receptors in Neurological Diseases With Motor Symptoms: Targets for Therapy.Coiled-coil structure-dependent interactions between polyQ proteins and Foxo lead to dendrite pathology and behavioral defects
P2860
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
@zh-cn
2017年学术文章
@zh-hans
2017年学术文章
@zh-my
2017年学术文章
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2017年學術文章
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2017年學術文章
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name
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@en
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@nl
type
label
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@en
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@nl
prefLabel
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@en
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@nl
P2093
P2860
P356
P1476
Polyglutamine spinocerebellar ataxias - from genes to potential treatments.
@en
P2093
H Brent Clark
Henry L Paulson
Vikram G Shakkottai
P2860
P2888
P304
P356
10.1038/NRN.2017.92
P407
P50
P577
2017-08-17T00:00:00Z