Renal Tubular Ubiquitin-Protein Ligase NEDD4-2 Is Required for Renal Adaptation during Long-Term Potassium Depletion.
about
Deletion of Nedd4-2 results in progressive kidney disease in mice.Dietary sodium induces a redistribution of the tubular metabolic workload.The endosomal trafficking factors CORVET and ESCRT suppress plasma membrane residence of the renal outer medullary potassium channel (ROMK).Potassium-Regulated Distal Tubule WNK Bodies are Kidney-Specific WNK1 Dependent.
P2860
Renal Tubular Ubiquitin-Protein Ligase NEDD4-2 Is Required for Renal Adaptation during Long-Term Potassium Depletion.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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name
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@en
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@nl
type
label
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@en
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@nl
prefLabel
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@en
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@nl
P2093
P2860
P356
P1476
Renal Tubular Ubiquitin-Protei ...... Long-Term Potassium Depletion.
@en
P2093
Ankita Roy
Arohan R Subramanya
Denis Basquin
Lama Al-Qusairi
Marc P Maillard
Olivier Staub
Renuga Devi Rajaram
P2860
P304
P356
10.1681/ASN.2016070732
P577
2017-03-13T00:00:00Z