Ubiquitylation and control of renal Na+ balance and blood pressure - Test2 - elhamkhani - TriplyDB

Ubiquitylation and control of renal Na+ balance and blood pressure

Ubiquitylation and control of renal Na+ balance and blood pressure

http://www.wikidata.org/entity/Q26863205

about

CPT-cGMP Is A New Ligand of Epithelial Sodium Channels Inhibition of ENaC by endothelin-1 Protein trafficking defects in inherited kidney diseases Understanding renal physiology leads to therapeutic advances in renal disease Enhanced expression of epithelial sodium channels causes salt-induced hypertension in mice through inhibition of the α2-isoform of Na+, K+-ATPase A Systems Level Analysis of Vasopressin-mediated Signaling Networks in Kidney Distal Convoluted Tubule Cells.Dietary potassium and the renal control of salt balance and blood pressure.Actions of Quercetin, a Polyphenol, on Blood Pressure.The thiazide sensitive sodium chloride co-transporter NCC is modulated by site-specific ubiquitylation.Physiology in perspective: the life we take for granted.Specific Palmitoyltransferases Associate with and Activate the Epithelial Sodium Channel.Renal Tubular Ubiquitin-Protein Ligase NEDD4-2 Is Required for Renal Adaptation during Long-Term Potassium Depletion.

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Ubiquitylation and control of renal Na+ balance and blood pressure

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description

2014 nî lūn-bûn

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2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2014 թվականի հունվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Ubiquitylation and control of renal Na+ balance and blood pressure

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Ubiquitylation and control of renal Na+ balance and blood pressure

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Ubiquitylation and control of renal Na+ balance and blood pressure

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Ubiquitylation and control of renal Na+ balance and blood pressure

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Ubiquitylation and control of renal Na+ balance and blood pressure

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PHYSIOL.00021.2013

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Ubiquitylation and control of renal Na+ balance and blood pressure

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C. Ronzaud

http://www.w3.org/2001/XMLSchema#string

O. Staub

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The Nedd4-like protein KIAA0439 is a potential regulator of the epithelial sodium channel

The thiazide-sensitive Na-Cl cotransporter is regulated by a WNK kinase signaling complex

KLHL3 mutations cause familial hyperkalemic hypertension by impairing ion transport in the distal nephron

Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

Impaired KLHL3-mediated ubiquitination of WNK4 causes human hypertension

Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4

Regulation of stability and function of the epithelial Na+ channel (ENaC) by ubiquitination.

Phosphorylation of Nedd4-2 by Sgk1 regulates epithelial Na(+) channel cell surface expression

Molecular physiology of the thiazide-sensitive sodium-chloride cotransporter

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16-26

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scholarly article

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10.1152/PHYSIOL.00021.2013

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1

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29

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2014-01-01T00:00:00Z

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