Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits? - Test2 - elhamkhani - TriplyDB

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

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Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

http://www.wikidata.org/entity/Q54114687

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh-hant

name

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@en

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

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type

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Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@en

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@nl

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Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@en

Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@nl

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FNEUR.2018.00228

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Frontiers in Neurology

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Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?

@en

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Jianzhong Yu

http://www.w3.org/2001/XMLSchema#string

Yulan Xiong

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P2860

Pathogenic LRRK2 negatively regulates microRNA-mediated translational repression

Ribosomal protein s15 phosphorylation mediates LRRK2 neurodegeneration in Parkinson's disease

LRRK2 controls an EndoA phosphorylation cycle in synaptic endocytosis

α-Synuclein interacts with the switch region of Rab8a in a Ser129 phosphorylation-dependent manner

Alpha-synuclein acts in the nucleus to inhibit histone acetylation and promote neurotoxicity

The retromer complex influences Wnt secretion by recycling wntless from endosomes to the trans-Golgi network

Wingless secretion requires endosome-to-Golgi retrieval of Wntless/Evi/Sprinter by the retromer complex

Gaucher disease glucocerebrosidase and α-synuclein form a bidirectional pathogenic loop in synucleinopathies

ArfGAP1 is a GTPase activating protein for LRRK2: reciprocal regulation of ArfGAP1 by LRRK2

Pre-fibrillar alpha-synuclein variants with impaired beta-structure increase neurotoxicity in Parkinson's disease models

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228

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scholarly article

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10.3389/FNEUR.2018.00228

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9

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2018-04-09T00:00:00Z

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29686647

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5900015

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