Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
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Inflammasomes: too big to missIL-18 and Cutaneous Inflammatory DiseasesImmune Homeostasis in Epithelial Cells: Evidence and Role of Inflammasome Signaling ReviewedActivated protein C: A regulator of human skin epidermal keratinocyte functionInflammasome activation of IL-1 family mediators in response to cutaneous photodamageAn Update on PYRIN Domain-Containing Pattern Recognition Receptors: From Immunity to PathologyTargeting the redox balance in inflammatory skin conditionsControl of innate and adaptive immunity by the inflammasomeBeyond pattern recognition: NOD-like receptors in dendritic cells.Mechanisms of chemical-induced innate immunity in allergic contact dermatitis.Novel concept of iSALT (inducible skin-associated lymphoid tissue) in the elicitation of allergic contact dermatitisNatural Compounds as Regulators of NLRP3 Inflammasome-Mediated IL-1β ProductionMonobenzone-induced depigmentation: from enzymatic blockade to autoimmunityNLRs, inflammasomes, and viral infectionCOPs and POPs: modulators of inflammasome activityHapten-induced contact hypersensitivity, autoimmune reactions, and tumor regression: plausibility of mediating antitumor immunityActivation of inflammasomes requires intracellular redistribution of the apoptotic speck-like protein containing a caspase recruitment domainDifferential splicing of the apoptosis-associated speck like protein containing a caspase recruitment domain (ASC) regulates inflammasomesThe NALP1 inflammasome controls cytokine production and nociception in a rat fracture model of complex regional pain syndromeCutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivityContact sensitizers induce skin inflammation via ROS production and hyaluronic acid degradationThe inflammasome NLRs in immunity, inflammation, and associated diseasesAnchoring molecular mechanisms to the adverse outcome pathway for skin sensitization: Analysis of existing data.Constitutively active inflammasome in human melanoma cells mediating autoinflammation via caspase-1 processing and secretion of interleukin-1betaLight, including ultraviolet.Skin-resident T cells sense ultraviolet radiation-induced injury and contribute to DNA repair.Challenging a Misnomer? The Role of Inflammatory Pathways in Inflammatory Breast CancerAlarmins, inflammasomes and immunity.Caspase-1 modulates incisional sensitization and inflammationSkin immune sentinels in health and disease.Caspase functions in cell death and disease.Toll-like receptor 4 has an essential role in early skin wound healing.Dendritic cell IL-1α and IL-1β are polyubiquitinated and degraded by the proteasome.Redox regulation of NLRP3 inflammasomes: ROS as trigger or effector?Nod-like receptor protein-3 inflammasome plays an important role during early stages of wound healing.Francisella infection triggers activation of the AIM2 inflammasome in murine dendritic cellsInflammasomes in cardiovascular diseases.Effects of Ixeris dentata water extract and caffeic acid on allergic inflammation in vivo and in vitro.IL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity responseAllergic contact dermatitis: epidemiology, molecular mechanisms, in vitro methods and regulatory aspects. Current knowledge assembled at an international workshop at BfR, Germany.
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Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
description
article
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im April 2007 veröffentlichter wissenschaftlicher Artikel
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wetenschappelijk artikel
@nl
наукова стаття, опублікована в серпні 2007
@uk
name
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@en
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@nl
type
label
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@en
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@nl
prefLabel
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@en
Activation of the IL-1β-Processing Inflammasome Is Involved in Contact Hypersensitivity
@nl
P2093
P50
P356
P1476
Activation of the IL-1beta-pro ...... ed in contact hypersensitivity
@en
P2093
Emmanuel Contassot
Hideki Watanabe
Jean A Kummer
Jürg Tschopp
Olivier Gaide
Stéphanie Roques
P2888
P304
P356
10.1038/SJ.JID.5700819
P407
P577
2007-04-12T00:00:00Z