CCAAT/enhancer binding protein epsilon is preferentially up-regulated during granulocytic differentiation and its functional versatility is determined by alternative use of promoters and differential splicing - Wikidata - thesis-toulmin - TriplyDB

CCAAT/enhancer binding protein epsilon is preferentially up-regulated during granulocytic differentiation and its functional versatility is determined by alternative use of promoters and differential splicing

CCAAT/enhancer binding protein epsilon is preferentially up-regulated during granulocytic differentiation and its functional versatility is determined by alternative use of promoters and differential splicing

http://www.wikidata.org/entity/Q24682820

about

Human resistin is a systemic immune-derived proinflammatory cytokine targeting both leukocytes and adipocytes CCAAT/enhancer-binding proteins: structure, function and regulation PRAM-1 is required for optimal integrin-dependent neutrophil function Upstream open reading frames: molecular switches in (patho)physiology Neutrophil-specific granule deficiency results from a novel mutation with loss of function of the transcription factor CCAAT/enhancer binding protein epsilon CCAAT/enhancer binding protein β in relation to ER stress, inflammation, and metabolic disturbances PRAM-1 is a novel adaptor protein regulated by retinoic acid (RA) and promyelocytic leukemia (PML)-RA receptor alpha in acute promyelocytic leukemia cells Novel combinatorial interactions of GATA-1, PU.1, and C/EBPepsilon isoforms regulate transcription of the gene encoding eosinophil granule major basic protein Inhibition of monocytic differentiation by phosphorylation-deficient Stat1 is associated with impaired expression of Stat2, ICSBP/IRF8 and C/EBPepsilon Granulocyte colony-stimulating factor receptors on CD34++ cells in patients with myelodysplastic syndrome (MDS) and MDS-acute myeloid leukemia N-terminal region of CCAAT/enhancer-binding protein epsilon is critical for cell cycle arrest, apoptosis, and functional maturation during myeloid differentiation C/EBPepsilon is a myeloid-specific activator of cytokine, chemokine, and macrophage-colony-stimulating factor receptor genes Spi-B can functionally replace PU.1 in myeloid but not lymphoid development Genomics of racial and ethnic disparities in childhood acute lymphoblastic leukemia.The C/EBP family of transcription factors in the liver and other organs.C/EBPepsilon directs granulocytic-vs-monocytic lineage determination and confers chemotactic function via Hlx.CCAAT/enhancer binding protein alpha is a regulatory switch sufficient for induction of granulocytic development from bipotential myeloid progenitors CCAAT/enhancer binding protein epsilon is a potential retinoid target gene in acute promyelocytic leukemia treatment.Dichotomy of AML1-ETO functions: growth arrest versus block of differentiation Alternative splicing of the human cyclin D-binding Myb-like protein (hDMP1) yields a truncated protein isoform that alters macrophage differentiation patterns.Identification of a responsible promoter region and a key transcription factor, CCAAT/enhancer-binding protein epsilon, for up-regulation of PHGPx in HL60 cells stimulated with TNF alpha.C/EBPε mediates nicotinamide-enhanced clearance of Staphylococcus aureus in mice.Global Identification of EVI1 Target Genes in Acute Myeloid Leukemia.CCAAT/enhancer-binding protein beta: its role in breast cancer and associations with receptor tyrosine kinases.The C/EBPbeta transcription factor regulates epithelial cell proliferation and differentiation in the mammary gland Negative regulation of STAT92E by an N-terminally truncated STAT protein derived from an alternative promoter site.PML-retinoic acid receptor alpha inhibits PML IV enhancement of PU.1-induced C/EBPepsilon expression in myeloid differentiation.Site-specific CpG methylation in the CCAAT/enhancer binding protein delta (CEBPδ) CpG island in breast cancer is associated with metastatic relapse C/EBPγ deregulation results in differentiation arrest in acute myeloid leukemia.C/EBP epsilon mediates myeloid differentiation and is regulated by the CCAAT displacement protein (CDP/cut).Metabolic role of dipeptidyl peptidase 4 (DPP4) in primary human (pre)adipocytes.Impaired granulopoiesis, myelodysplasia, and early lethality in CCAAT/enhancer binding protein epsilon-deficient mice.Human C/EBP-epsilon activator and repressor isoforms differentially reprogram myeloid lineage commitment and differentiation Identification of neuronal target genes for CCAAT/enhancer binding proteins.CCAAT/enhancer-binding proteins and the pathogenesis of retrovirus infection.Expression of different functional isoforms in haematopoiesis.CCAAT/enhancer-binding protein activates the CD14 promoter and mediates transforming growth factor beta signaling in monocyte development.Eicosapentaenoic acid demethylates a single CpG that mediates expression of tumor suppressor CCAAT/enhancer-binding protein delta in U937 leukemia cells Wnt activation and alternative promoter repression of LEF1 in colon cancer.Interaction between CCAAT/enhancer binding protein and cyclic AMP response element binding protein 1 regulates human immunodeficiency virus type 1 transcription in cells of the monocyte/macrophage lineage.

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P2860

CCAAT/enhancer binding protein epsilon is preferentially up-regulated during granulocytic differentiation and its functional versatility is determined by alternative use of promoters and differential splicing

http://www.wikidata.org/entity/Q24682820

description

1997 nî lūn-bûn

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1997 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

1997 թվականի հունիսին հրատարակված գիտական հոդված

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1997年の論文

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1997年論文

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1997年論文

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1997年論文

@zh-hk

1997年論文

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1997年論文

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1997年论文

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name

CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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type

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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CCAAT/enhancer binding protein ...... ters and differential splicing

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PNAS.94.12.6462

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Proceedings of the National Academy of Sciences of the United States of America

P1476

CCAAT/enhancer binding protein ...... ters and differential splicing

@en

P2093

G D Kim

http://www.w3.org/2001/XMLSchema#string

H S Radomska

http://www.w3.org/2001/XMLSchema#string

J Lekstrom-Himes

http://www.w3.org/2001/XMLSchema#string

K G Xanthopoulos

http://www.w3.org/2001/XMLSchema#string

L T Smith

http://www.w3.org/2001/XMLSchema#string

R Yamanaka

http://www.w3.org/2001/XMLSchema#string

P2860

Ig/EBP (C/EBP gamma) is a transdominant negative inhibitor of C/EBP family transcriptional activators

A novel human CCAAT/enhancer binding protein gene, C/EBPepsilon, is expressed in cells of lymphoid and myeloid lineages and is localized on chromosome 14q11.2 close to the T-cell receptor alpha/delta locus

The rat hepatic leukemia factor (HLF) gene encodes two transcriptional activators with distinct circadian rhythms, tissue distributions and target preferences

An analysis of vertebrate mRNA sequences: intimations of translational control

Cloning of the novel human myeloid-cell-specific C/EBP-epsilon transcription factor

Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells

More is better: activators and repressors from the same gene

A family of C/EBP-related proteins capable of forming covalently linked leucine zipper dimers in vitro

IL-6DBP, a nuclear protein involved in interleukin-6 signal transduction, defines a new family of leucine zipper proteins related to C/EBP

CCAAT/enhancer-binding protein mRNA is translated into multiple proteins with different transcription activation potentials

P304

6462-6467

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scholarly article

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10.1073/PNAS.94.12.6462

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12

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94

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14042029

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9177240

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21072

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