Enhanced osteoclastic resorption and responsiveness to mechanical load in gap junction deficient bone. - Wikidata - thesis-toulmin - TriplyDB

Enhanced osteoclastic resorption and responsiveness to mechanical load in gap junction deficient bone.

Enhanced osteoclastic resorption and responsiveness to mechanical load in gap junction deficient bone.

http://www.wikidata.org/entity/Q34012735

about

Gap junctional regulation of signal transduction in bone cells The interaction of biological factors with mechanical signals in bone adaptation: recent developments Functions of the osteocyte network in the regulation of bone mass Osteocytes: master orchestrators of bone Apoptotic osteocytes and the control of targeted bone resorption Connexin 43 hemichannels and intracellular signaling in bone cells Osteogenic gene transcription is regulated via gap junction-mediated cell-cell communication Physiological mechanisms and therapeutic potential of bone mechanosensing.Age-related changes in gap junctional intercellular communication in osteoblastic cells In situ intracellular calcium oscillations in osteocytes in intact mouse long bones under dynamic mechanical loading.High glucose alters Cx43 expression and gap junction intercellular communication in retinal Müller cells: promotes Müller cell and pericyte apoptosis.Skeletal adaptation to intramedullary pressure-induced interstitial fluid flow is enhanced in mice subjected to targeted osteocyte ablation.Enhanced periosteal and endocortical responses to axial tibial compression loading in conditional connexin43 deficient mice A new method to investigate how mechanical loading of osteocytes controls osteoblasts A novel autosomal recessive GJA1 missense mutation linked to Craniometaphyseal dysplasia Inhibition of GSK-3β rescues the impairments in bone formation and mechanical properties associated with fracture healing in osteoblast selective connexin 43 deficient mice.Connexin 43 channels are essential for normal bone structure and osteocyte viability Up-regulation of BMP2/4 signaling increases both osteoblast-specific marker expression and bone marrow adipogenesis in Gja1Jrt/+ stromal cell cultures.Evidence for the role of connexin 43-mediated intercellular communication in the process of intracortical bone resorption via osteocytic osteolysis Cx43 and mechanotransduction in bone.Deletion of connexin43 in osteoblasts/osteocytes leads to impaired muscle formation in mice.Connexin 43 deficiency desensitizes bone to the effects of mechanical unloading through modulation of both arms of bone remodeling Connexins and pannexins in the skeleton: gap junctions, hemichannels and more Role of connexins and pannexins during ontogeny, regeneration, and pathologies of bone Bisphosphonates improve trabecular bone mass and normalize cortical thickness in ovariectomized, osteoblast connexin43 deficient mice.Defective cancellous bone structure and abnormal response to PTH in cortical bone of mice lacking Cx43 cytoplasmic C-terminus domain.Beyond gap junctions: Connexin43 and bone cell signaling Impact of Gap Junctional Intercellular Communication on MLO-Y4 Sclerostin and Soluble Factor Expression.Connexins in the skeleton Osteoblast and osteocyte-specific loss of Connexin43 results in delayed bone formation and healing during murine fracture healing The regulation of runt-related transcription factor 2 by fibroblast growth factor-2 and connexin43 requires the inositol polyphosphate/protein kinase Cδ cascade Absence of Cx43 selectively from osteocytes enhances responsiveness to mechanical force in mice Connexin 43 deficiency attenuates loss of trabecular bone and prevents suppression of cortical bone formation during unloading.Deletion of Cx43 from osteocytes results in defective bone material properties but does not decrease extrinsic strength in cortical bone The osteocyte: an endocrine cell ... and more.Osteocytic connexin hemichannels suppress breast cancer growth and bone metastasis.Glucocorticoid impairs cell-cell communication by autophagy-mediated degradation of connexin 43 in osteocytes.Connexin43 modulates post-natal cortical bone modeling and mechano-responsiveness.Osteocyte-driven bone remodeling.High bone mass in mice lacking Cx37 because of defective osteoclast differentiation.

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Enhanced osteoclastic resorption and responsiveness to mechanical load in gap junction deficient bone.

http://www.wikidata.org/entity/Q34012735

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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type

label

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

@ast

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

@en

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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prefLabel

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

@ast

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

@en

Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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JOURNAL.PONE.0023516

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Enhanced osteoclastic resorpti ...... n gap junction deficient bone.

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Andrew Davison

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Emmanuel M Paul

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Henry J Donahue

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Neil Sharkey

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Sundar Srinivasan

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Ted S Gross

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Vikram Sathyendra

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Yue Zhang

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P2860

Connexin 43 (GJA1) mutations cause the pleiotropic phenotype of oculodentodigital dysplasia

Mechanical stimulation of bone in vivo reduces osteocyte expression of Sost/sclerostin

Connexin46 is retained as monomers in a trans-Golgi compartment of osteoblastic cells

The P2X7 nucleotide receptor mediates skeletal mechanotransduction

Adjusting data to body size: a comparison of methods as applied to quantitative trait loci analysis of musculoskeletal phenotypes.

Gap junctions regulate extracellular signal-regulated kinase signaling to affect gene transcription.

Differentiation of human fetal osteoblastic cells and gap junctional intercellular communication.

Gap junctions and biophysical regulation of bone cell differentiation.

Connexin43 mediates direct intercellular communication in human osteoblastic cell networks

Oscillating fluid flow activation of gap junction hemichannels induces ATP release from MLO-Y4 osteocytes.

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10.1371/JOURNAL.PONE.0023516

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8

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Sarah K. Bronson

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51625030

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21897843

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3163577

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