N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor
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siRNA knockdown of GPR18 receptors in BV-2 microglia attenuates N-arachidonoyl glycine-induced cell migrationInternational Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligandsTaming THC: potential cannabis synergy and phytocannabinoid-terpenoid entourage effectsN-Arachidonyl glycine does not activate G protein-coupled receptor 18 signaling via canonical pathwaysThe Endocannabinoid System as a Therapeutic Target in GlaucomaExpression and Function of the Endocannabinoid System in the Retina and the Visual BrainAlternative targets within the endocannabinoid system for future treatment of gastrointestinal diseasesThe cannabinoid acids, analogs and endogenous counterpartsThe endocannabinoid/endovanilloid N-arachidonoyl dopamine (NADA) and synthetic cannabinoid WIN55,212-2 abate the inflammatory activation of human endothelial cellsΔ(9) -Tetrahydrocannabinol and N-arachidonyl glycine are full agonists at GPR18 receptors and induce migration in human endometrial HEC-1B cellsInternational Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid receptors and their ligands: beyond CB₁ and CB₂GPR55, a G-protein coupled receptor for lysophosphatidylinositol, plays a role in motor coordination.Characterization of non-olfactory GPCRs in human sperm with a focus on GPR18Long-term consequences of perinatal fatty acid amino hydrolase inhibition.Minireview: recent developments in the physiology and pathology of the lysophosphatidylinositol-sensitive receptor GPR55.CB2 receptor activation inhibits melanoma cell transmigration through the blood-brain barrier.Activation of GPR18 by cannabinoid compounds: a tale of biased agonism.The non-psychoactive plant cannabinoid, cannabidiol affects cholesterol metabolism-related genes in microglial cells.Neuronal nitric oxide synthase-dependent elevation in adiponectin in the rostral ventrolateral medulla underlies g protein-coupled receptor 18-mediated hypotension in conscious rats.The atypical cannabinoid O-1602 protects against experimental colitis and inhibits neutrophil recruitmentA GPR18-based signalling system regulates IOP in murine eye.N-arachidonyl-glycine modulates synaptic transmission in superficial dorsal hornAbnormal cannabidiol attenuates experimental colitis in mice, promotes wound healing and inhibits neutrophil recruitment.Resolvin D2 Enhances Postischemic Revascularization While Resolving Inflammation.Microarray and pathway analysis reveal distinct mechanisms underlying cannabinoid-mediated modulation of LPS-induced activation of BV-2 microglial cellsCannabidiol as an emergent therapeutic strategy for lessening the impact of inflammation on oxidative stress.Identification of the GPR55 antagonist binding site using a novel set of high-potency GPR55 selective ligands.The therapeutic potential of cannabinoids for movement disorders.Endocannabinoids and traumatic brain injuryCannabidiol inhibits pathogenic T cells, decreases spinal microglial activation and ameliorates multiple sclerosis-like disease in C57BL/6 mice.High-resolution metabolomics to discover potential parasite-specific biomarkers in a Plasmodium falciparum erythrocytic stage culture systemNovel endogenous N-acyl amides activate TRPV1-4 receptors, BV-2 microglia, and are regulated in brain in an acute model of inflammation.Resolution of inflammation by N-arachidonoylglycine.The therapeutic potential of orphan GPCRs, GPR35 and GPR55.Alkylindole-sensitive receptors modulate microglial cell migration and proliferation.Low-variance RNAs identify Parkinson's disease molecular signature in bloodPrimary Macrophage Chemotaxis Induced by Cannabinoid Receptor 2 Agonists Occurs Independently of the CB2 Receptor.N-Amino acid linoleoyl conjugates: anti-inflammatory activitiesAnandamide, Acting via CB2 Receptors, Alleviates LPS-Induced Neuroinflammation in Rat Primary Microglial Cultures.Pathogenesis of Systemic Sclerosis.
P2860
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P2860
N-arachidonoyl glycine, an abundant endogenous lipid, potently drives directed cellular migration through GPR18, the putative abnormal cannabidiol receptor
description
2010 nî lūn-bûn
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2010 թուականին հրատարակուած գիտական յօդուած
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2010 թվականին հրատարակված գիտական հոդված
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2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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name
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@ast
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en-gb
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@nl
type
label
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@ast
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en-gb
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@nl
prefLabel
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@ast
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en-gb
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@nl
P2093
P2860
P3181
P356
P1433
P1476
N-arachidonoyl glycine, an abu ...... abnormal cannabidiol receptor
@en
P2093
Ana Juknat
Heather B Bradshaw
J Michael Walker
Neta Rimmerman
Sherry S J Hu
P2860
P2888
P3181
P356
10.1186/1471-2202-11-44
P407
P577
2010-01-01T00:00:00Z
P5875
P6179
1045430186