Disrupted axo-glial junctions result in accumulation of abnormal mitochondria at nodes of ranvier
about
A putative functional role for oligodendrocytes in mood regulationIn vivo deletion of immunoglobulin domains 5 and 6 in neurofascin (Nfasc) reveals domain-specific requirements in myelinated axonsCaspr and caspr2 are required for both radial and longitudinal organization of myelinated axons.Electron tomographic analysis of cytoskeletal cross-bridges in the paranodal region of the node of Ranvier in peripheral nerves.Electron tomography of paranodal septate-like junctions and the associated axonal and glial cytoskeletons in the central nervous system.Activity-dependent regulation of mitochondrial motility by calcium and Na/K-ATPase at nodes of Ranvier of myelinated nerves.No effect of genetic deletion of contactin-associated protein (CASPR) on axonal orientation and synaptic plasticity.Spatiotemporal ablation of myelinating glia-specific neurofascin (Nfasc NF155) in mice reveals gradual loss of paranodal axoglial junctions and concomitant disorganization of axonal domains.Multivesicular bodies in neurons: distribution, protein content, and trafficking functions.Mitochondrial configurations in peripheral nerve suggest differential ATP production.The cytoskeletal adaptor protein band 4.1B is required for the maintenance of paranodal axoglial septate junctions in myelinated axons.Activated microglia mediate axoglial disruption that contributes to axonal injury in multiple sclerosisA novel and robust conditioning lesion induced by ethidium bromide.The 4.1B cytoskeletal protein regulates the domain organization and sheath thickness of myelinated axons.Matching mitochondria to metabolic needs at nodes of Ranvier.Concepts for regulation of axon integrity by enwrapping glia.Schwann cell myelinationQuantitative analysis of multivesicular bodies (MVBs) in the hypoglossal nerve: evidence that neurotrophic factors do not use MVBs for retrograde axonal transportMitochondrial Mg(2+) homeostasis decides cellular energy metabolism and vulnerability to stress.Cytoskeletal transition at the paranodes: the Achilles' heel of myelinated axons.Structure and function of the contactin-associated protein family in myelinated axons and their relationship with nerve diseases.Disruption of paranodal axo-glial interaction and/or absence of sulfatide causes irregular type I inositol 1,4,5-trisphosphate receptor deposition in cerebellar Purkinje neuron axons.Disruption of White Matter Integrity by Chronic Cerebral Hypoperfusion in Alzheimer's Disease Mouse Model.
P2860
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P2860
Disrupted axo-glial junctions result in accumulation of abnormal mitochondria at nodes of ranvier
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2006 nî lūn-bûn
@nan
2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի օգոստոսին հրատարակված գիտական հոդված
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2006年の論文
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2006年論文
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2006年論文
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2006年論文
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2006年論文
@zh-mo
2006年論文
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2006年论文
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name
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@ast
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@en
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@nl
type
label
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@ast
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@en
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@nl
prefLabel
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@ast
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@en
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@nl
P2860
P3181
P1433
P1476
Disrupted axo-glial junctions ...... tochondria at nodes of ranvier
@en
P2093
Steven Einheber
P2860
P304
P3181
P356
10.1017/S1740925X06000275
P407
P577
2006-08-01T00:00:00Z