Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineage - Wikidata - wikimedia - TriplyDB

Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineage

Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineage

http://www.wikidata.org/entity/Q27312357

about

Silencing Mist1 Gene Expression Is Essential for Recovery from Acute Pancreatitis Utilizing past and present mouse systems to engineer more relevant pancreatic cancer models Molecular imaging in pancreatic cancer--a roadmap for therapeutic decisions.The transcription factor GLI1 modulates the inflammatory response during pancreatic tissue remodeling Kras as a key oncogene and therapeutic target in pancreatic cancer Epithelial Notch signaling is a limiting step for pancreatic carcinogenesis.Dicer regulates differentiation and viability during mouse pancreatic cancer initiation Clinical impact of preoperative acute pancreatitis in patients who undergo pancreaticoduodenectomy for periampullary tumors Characterization of Mouse Models of Early Pancreatic Lesions Induced by Alcohol and Chronic Pancreatitis Bmi1 is required for the initiation of pancreatic cancer through an Ink4a-independent mechanism.Genetically engineered mouse models of pancreatic cancer.Nestin delineates pancreatic cancer stem cells in metastatic foci of NOD/Shi-scid IL2Rγ(null) (NOG) mice.The burning question: why is smoking a risk factor for pancreatic cancer?Acinar cell plasticity and development of pancreatic ductal adenocarcinoma.Neuroplastic changes occur early in the development of pancreatic ductal adenocarcinoma.Canonical wnt signaling is required for pancreatic carcinogenesis Maintenance of acinar cell organization is critical to preventing Kras-induced acinar-ductal metaplasia.Intracellular HMGB1 as a novel tumor suppressor of pancreatic cancer.Yes-associated protein mediates immune reprogramming in pancreatic ductal adenocarcinoma.CD4+ T lymphocyte ablation prevents pancreatic carcinogenesis in mice.Duct- and Acinar-Derived Pancreatic Ductal Adenocarcinomas Show Distinct Tumor Progression and Marker Expression.Malnutrition in Pancreatic Ductal Adenocarcinoma (PDA): Dietary Pancreatic Enzymes Improve Short-Term Health but Stimulate Tumor Growth.Nr5a2 heterozygosity sensitises to, and cooperates with, inflammation in KRas(G12V)-driven pancreatic tumourigenesis.Distribution of the cytoskeletal protein, Nestin, in acute leukemia.

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Acute pancreatitis accelerates initiation and progression to pancreatic cancer in mice expressing oncogenic Kras in the nestin cell lineage

http://www.wikidata.org/entity/Q27312357

description

2011 nî lūn-bûn

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2011 թուականին հրատարակուած գիտական յօդուած

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2011 թվականին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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type

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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JOURNAL.PONE.0027725

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Acute pancreatitis accelerates ...... ras in the nestin cell lineage

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Alison L Young

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Catherine Carrière

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Daniel S Longnecker

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Jason R Gunn

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Murray Korc

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P2860

Distant metastasis occurs late during the genetic evolution of pancreatic cancer

Generalized lacZ expression with the ROSA26 Cre reporter strain

Origin of exocrine pancreatic cells from nestin-positive precursors in developing mouse pancreas

Disruption of the glucocorticoid receptor gene in the nervous system results in reduced anxiety

The patterns and dynamics of genomic instability in metastatic pancreatic cancer

Direct evidence for the pancreatic lineage: NGN3+ cells are islet progenitors and are distinct from duct progenitors

Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse

Beta-catenin blocks Kras-dependent reprogramming of acini into pancreatic cancer precursor lesions in mice.

Epithelial tissues have varying degrees of susceptibility to Kras(G12D)-initiated tumorigenesis in a mouse model.

Adult pancreatic acinar cells give rise to ducts but not endocrine cells in response to growth factor signaling.

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e27725

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scholarly article

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10.1371/JOURNAL.PONE.0027725

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11

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2011-01-01T00:00:00Z

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3225359

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