Transgenic incorporation of skeletal TnT into cardiac myofilaments blunts PKC-mediated depression of force
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Gene regulation, alternative splicing, and posttranslational modification of troponin subunits in cardiac development and adaptation: a focused reviewRaf-1: a novel cardiac troponin T kinaseASK1 associates with troponin T and induces troponin T phosphorylation and contractile dysfunction in cardiomyocytesPhosphorylation of protein kinase C sites Ser42/44 decreases Ca(2+)-sensitivity and blunts enhanced length-dependent activation in response to protein kinase A in human cardiomyocytesProtein kinase C alpha and epsilon phosphorylation of troponin and myosin binding protein C reduce Ca2+ sensitivity in human myocardiumProtein kinase C epsilon induces systolic cardiac failure marked by exhausted inotropic reserve and intact Frank-Starling mechanismWhy does troponin I have so many phosphorylation sites? Fact and fancyTroponin T isoforms and posttranscriptional modifications: Evolution, regulation and functionCapZ dynamics are altered by endothelin-1 and phenylephrine via PIP2- and PKC-dependent mechanisms.Temporal and mutation-specific alterations in Ca2+ homeostasis differentially determine the progression of cTnT-related cardiomyopathies in murine models.Molecular actions of drugs that sensitize cardiac myofilaments to Ca2+.Ceramide-mediated depression in cardiomyocyte contractility through PKC activation and modulation of myofilament protein phosphorylationChronic coexistence of two troponin T isoforms in adult transgenic mouse cardiomyocytes decreased contractile kinetics and caused dilatative remodelingThe N-terminal extension of cardiac troponin T stabilizes the blocked state of cardiac thin filamentTNNT1, TNNT2, and TNNT3: Isoform genes, regulation, and structure-function relationships.The role of protein kinase C-mediated phosphorylation of sarcomeric proteins in the heart-detrimental or beneficial?Troponin I serines 43/45 and regulation of cardiac myofilament function.alpha-Adrenergic response and myofilament activity in mouse hearts lacking PKC phosphorylation sites on cardiac TnI.Protein kinase C and A sites on troponin I regulate myofilament Ca2+ sensitivity and ATPase activity in the mouse myocardium.Increase in tension-dependent ATP consumption induced by cardiac troponin T mutation.Hypertrophic cardiomyopathy mutation in cardiac troponin T (R95H) attenuates length-dependent activation in guinea pig cardiac muscle fibers.
P2860
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P2860
Transgenic incorporation of skeletal TnT into cardiac myofilaments blunts PKC-mediated depression of force
description
2001 nî lūn-bûn
@nan
2001 թուականի Մարտին հրատարակուած գիտական յօդուած
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2001 թվականի մարտին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年论文
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name
Transgenic incorporation of sk ...... C-mediated depression of force
@ast
Transgenic incorporation of sk ...... C-mediated depression of force
@en
Transgenic incorporation of sk ...... C-mediated depression of force
@nl
type
label
Transgenic incorporation of sk ...... C-mediated depression of force
@ast
Transgenic incorporation of sk ...... C-mediated depression of force
@en
Transgenic incorporation of sk ...... C-mediated depression of force
@nl
prefLabel
Transgenic incorporation of sk ...... C-mediated depression of force
@ast
Transgenic incorporation of sk ...... C-mediated depression of force
@en
Transgenic incorporation of sk ...... C-mediated depression of force
@nl
P2093
P2860
P1476
Transgenic incorporation of sk ...... C-mediated depression of force
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.2001.280.3.H1011
P407
P577
2001-03-01T00:00:00Z