Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth - Wikidata - wikimedia - TriplyDB

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

http://www.wikidata.org/entity/Q28389589

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ICP0 dismantles microtubule networks in herpes simplex virus-infected cells The DNA-damage response in human biology and disease A viral E3 ligase targets RNF8 and RNF168 to control histone ubiquitination and DNA damage responses HSV-1 ICP0: paving the way for viral replication ICP0 inhibits the decrease of HSV amplicon-mediated transgene expression.HSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate Immunity Avian reovirus nonstructural protein p17-induced G(2)/M cell cycle arrest and host cellular protein translation shutoff involve activation of p53-dependent pathways.Human herpesvirus 6 suppresses T cell proliferation through induction of cell cycle arrest in infected cells in the G2/M phase.Activation of checkpoint kinase 2 is critical for herpes simplex virus type 1 replication in corneal epithelium Activation of H2AX and ATM in varicella-zoster virus (VZV)-infected cells is associated with expression of specific VZV genes Viral E3 ubiquitin ligase-mediated degradation of a cellular E3: viral mimicry of a cellular phosphorylation mark targets the RNF8 FHA domain.Viral manipulation of DNA repair and cell cycle checkpoints Inhibition of ataxia telangiectasia mutated (ATM) kinase suppresses herpes simplex virus type 1 (HSV-1) keratitis.Cell Cycle-Dependent Expression of Adeno-Associated Virus 2 (AAV2) Rep in Coinfections with Herpes Simplex Virus 1 (HSV-1) Gives Rise to a Mosaic of Cells Replicating either AAV2 or HSV-1.An Intrinsically Disordered Region of the DNA Repair Protein Nbs1 Is a Species-Specific Barrier to Herpes Simplex Virus 1 in Primates.H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication.Initiation of lytic DNA replication in Epstein-Barr virus: search for a common family mechanism.Therapeutic targeting of chemoresistant and recurrent glioblastoma stem cells with a proapoptotic variant of oncolytic herpes simplex virus.Physical interaction between the herpes simplex virus type 1 exonuclease, UL12, and the DNA double-strand break-sensing MRN complex.Dynamic Proteomics of Herpes Simplex Virus Infection.

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Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

http://www.wikidata.org/entity/Q28389589

description

2008 nî lūn-bûn

@nan

2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@ast

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@en

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@nl

type

label

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@ast

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@en

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@nl

prefLabel

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@ast

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@en

Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@nl

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P356

J.VIROL.2008.01.038

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Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth

@en

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David M Krisky

http://www.w3.org/2001/XMLSchema#string

Han Li

http://www.w3.org/2001/XMLSchema#string

Joseph C Glorioso

http://www.w3.org/2001/XMLSchema#string

Justus B Cohen

http://www.w3.org/2001/XMLSchema#string

Kiflai Bein

http://www.w3.org/2001/XMLSchema#string

Rajasekaran Baskaran

http://www.w3.org/2001/XMLSchema#string

P2860

Linkage of ATM to cell cycle regulation by the Chk2 protein kinase

A human homologue of the checkpoint kinase Cds1 directly inhibits Cdc25 phosphatase

Arsenite-induced Cdc25C Degradation is through the KEN-box and Ubiquitin-proteasome Pathway

The bovine herpesvirus 1 immediate-early protein (bICP0) associates with histone deacetylase 1 to activate transcription.

ATM activation by DNA double-strand breaks through the Mre11-Rad50-Nbs1 complex

Dephosphorylation and activation of a p34cdc2/cyclin B complex in vitro by human CDC25 protein

Components of the REST/CoREST/histone deacetylase repressor complex are disrupted, modified, and translocated in HSV-1-infected cells

ATR-mediated checkpoint pathways regulate phosphorylation and activation of human Chk1

Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint

Regulation of herpesvirus macromolecular synthesis. I. Cascade regulation of the synthesis of three groups of viral proteins

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13-23

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10.1016/J.VIROL.2008.01.038

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1

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375

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2008-05-25T00:00:00Z

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5529913

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2706573

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