Load regulates bone formation and Sclerostin expression through a TGFβ-dependent mechanism
about
Regulation of postnatal bone homeostasis by TGFβWhen 1+1>2: Nanostructured composites for hard tissue engineering applicationsDevelopments in sclerostin biology: regulation of gene expression, mechanisms of action, and physiological functions.Osteocyte expression of caspase-3, COX-2, IL-6 and sclerostin are spatially and temporally associated following stress fracture initiation.Effects of 18α-glycyrrhizin on TGF-β1/Smad signaling pathway in rats with carbon tetrachloride-induced liver fibrosis.Emerging targets in osteoarthritis therapy.Heavy Metal Ion Regulation of Gene Expression: MECHANISMS BY WHICH LEAD INHIBITS OSTEOBLASTIC BONE-FORMING ACTIVITY THROUGH MODULATION OF THE Wnt/β-CATENIN SIGNALING PATHWAY.The Wnt Inhibitor Sclerostin Is Up-regulated by Mechanical Unloading in Osteocytes in Vitro.Sclerostin Antibody Treatment Improves the Bone Phenotype of Crtap(-/-) Mice, a Model of Recessive Osteogenesis Imperfecta.Mechanobiology of TGFβ signaling in the skeleton.Role of TGF-β in breast cancer bone metastases.Tendon development and diseases.Sclerostin, Osteocytes, and Chronic Kidney Disease - Mineral Bone Disorder.Phenylethanol Glycosides from Cistanche tubulosa Suppress Hepatic Stellate Cell Activation and Block the Conduction of Signaling Pathways in TGF-β1/smad as Potential Anti-Hepatic Fibrosis Agents.Exploiting the WNT Signaling Pathway for Clinical Purposes.Loss of sclerostin promotes osteoarthritis in mice via β-catenin-dependent and -independent Wnt pathways.Osteocyte-Intrinsic TGF-β Signaling Regulates Bone Quality through Perilacunar/Canalicular Remodeling.Heterozygous deletion of both sclerostin (Sost) and connexin43 (Gja1) genes in mice is not sufficient to impair cortical bone modeling.Increased sclerostin associated with stress fracture of the third metacarpal bone in the Thoroughbred racehorse.Mechanical Force-induced TGFB1 Increases Expression of SOST/POSTN by hPDL Cells.Advanced Glycation End Product 3 (AGE3) Increases Apoptosis and the Expression of Sclerostin by Stimulating TGF-β Expression and Secretion in Osteocyte-Like MLO-Y4-A2 Cells.
P2860
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P2860
Load regulates bone formation and Sclerostin expression through a TGFβ-dependent mechanism
description
2013 nî lūn-bûn
@nan
2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
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name
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@ast
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@en
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@nl
type
label
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@ast
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@en
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@nl
prefLabel
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@ast
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@en
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@nl
P2860
P1433
P1476
Load regulates bone formation ...... ugh a TGFβ-dependent mechanism
@en
P2093
Daniel Nguyen
Jacqueline Nguyen
P2860
P304
P356
10.1371/JOURNAL.PONE.0053813
P407
P577
2013-01-01T00:00:00Z