The sensitivity of diffuse large B-cell lymphoma cell lines to histone deacetylase inhibitor-induced apoptosis is modulated by BCL-2 family protein activity
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Histone acetyltransferases and histone deacetylases in B- and T-cell development, physiology and malignancyLMP1+SLAMF1high cells are associated with drug resistance in Epstein-Barr virus-positive Farage cells.MYC expression in concert with BCL2 and BCL6 expression predicts outcome in Chinese patients with diffuse large B-cell lymphoma, not otherwise specified.A Bim-targeting strategy overcomes adaptive bortezomib resistance in myeloma through a novel link between autophagy and apoptosisSystematic analysis of time-series gene expression data on tumor cell-selective apoptotic responses to HDAC inhibitors.Novel HDAC inhibitors exhibit pre-clinical efficacy in lymphoma models and point to the importance of CDKN1A expression levels in mediating their anti-tumor responseTargeting Histone Deacetylases in Diseases: Where Are We?The histone deacetylase inhibitor trichostatin a decreases lymphangiogenesis by inducing apoptosis and cell cycle arrest via p21-dependent pathwaysPU.1-Bim axis is involved in Trichostatin A-induced apoptosis in murine pro-B lymphoma FL5.12 cells.Myc Induces miRNA-Mediated Apoptosis in Response to HDAC Inhibition in Hematologic Malignancies.Combination with vorinostat overcomes ABT-263 (navitoclax) resistance of small cell lung cancer.Vorinostat, a histone deacetylase (HDAC) inhibitor, promotes cell cycle arrest and re-sensitizes rituximab- and chemo-resistant lymphoma cells to chemotherapy agents.Anti-tumor activity of the combination of bendamustine with vorinostat in diffuse large B-cell lymphoma cells.Inhibition of Oncogenic Transcription Factor REL by the Natural Product Derivative Calafianin Monomer 101 Induces Proliferation Arrest and Apoptosis in Human B-Lymphoma Cell Lines.Histone acetyltransferase-deficient p300 mutants in diffuse large B cell lymphoma have altered transcriptional regulatory activities and are required for optimal cell growth.A novel approach to detect resistance mechanisms reveals FGR as a factor mediating HDAC inhibitor SAHA resistance in B-cell lymphoma.Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode.The Bcl-2 inhibitor Obatoclax overcomes resistance to histone deacetylase inhibitors SAHA and LBH589 as radiosensitizers in patient-derived glioblastoma stem-like cells.MicroRNA-21 regulates the viability and apoptosis of diffuse large B-cell lymphoma cells by upregulating B cell lymphoma-2.Vorinostat and metformin sensitize EGFR-TKI resistant NSCLC cells via BIM-dependent apoptosis induction.
P2860
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P2860
The sensitivity of diffuse large B-cell lymphoma cell lines to histone deacetylase inhibitor-induced apoptosis is modulated by BCL-2 family protein activity
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2013 nî lūn-bûn
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2013 թուականին հրատարակուած գիտական յօդուած
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2013 թվականին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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name
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@ast
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@en
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@nl
type
label
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@ast
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@en
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@nl
prefLabel
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@ast
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@en
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@nl
P2093
P2860
P3181
P1433
P1476
The sensitivity of diffuse lar ...... BCL-2 family protein activity
@en
P2093
Iosif Vardinogiannis
Ryan C Thompson
Thomas D Gilmore
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0062822
P407
P577
2013-01-01T00:00:00Z