Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury - Wikidata - wikimedia - TriplyDB

Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury

Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury

http://www.wikidata.org/entity/Q28571240

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Ribosomal Protein S6 Phosphorylation in the Nervous System: From Regulation to Function Linking traumatic brain injury to chronic traumatic encephalopathy: identification of potential mechanisms leading to neurofibrillary tangle development Rapamycin attenuates the development of posttraumatic epilepsy in a mouse model of traumatic brain injury The role of necroptosis in neurosurgical diseases Role of Akt-independent mTORC1 and GSK3β signaling in sublethal NMDA-induced injury and the recovery of neuronal electrophysiology and survival Developing Antiepileptogenic Drugs for Acquired Epilepsy: Targeting the Mammalian Target of Rapamycin (mTOR) Pathway.Role of Akt and mammalian target of rapamycin in functional outcome after concussive brain injury in mice Mechanisms of epileptogenesis: a convergence on neural circuit dysfunction.Role of the mTOR pathway in LPS-activated monocytes: influence of hypertonic saline Mammalian target of rapamycin (mTOR) inhibition as a potential antiepileptogenic therapy: From tuberous sclerosis to common acquired epilepsies Blockade of excitatory synaptogenesis with proximal dendrites of dentate granule cells following rapamycin treatment in a mouse model of temporal lobe epilepsy.Rapamycin suppresses mossy fiber sprouting but not seizure frequency in a mouse model of temporal lobe epilepsy.Genetic activation of mTORC1 signaling worsens neurocognitive outcome after traumatic brain injury.Frontier of epilepsy research - mTOR signaling pathway.Therapeutic role of mammalian target of rapamycin (mTOR) inhibition in preventing epileptogenesis.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.Rapamycin suppresses axon sprouting by somatostatin interneurons in a mouse model of temporal lobe epilepsy.Is there a critical period for mossy fiber sprouting in a mouse model of temporal lobe epilepsy?Xuefu Zhuyu decoction, a traditional Chinese medicine, provides neuroprotection in a rat model of traumatic brain injury via an anti-inflammatory pathway Targeting mTOR as a novel therapeutic strategy for traumatic CNS injuries.Modulation of the cAMP signaling pathway after traumatic brain injury.Decoding hippocampal signaling deficits after traumatic brain injury Beneficial Effects of Early mTORC1 Inhibition after Traumatic Brain Injury.Dysfunction of the mTOR pathway is a risk factor for Alzheimer's disease.Neuroprotective levels of IGF-1 exacerbate epileptogenesis after brain injury.Mammalian target of rapamycin (mTOR) inhibition: potential for antiseizure, antiepileptogenic, and epileptostatic therapy.A critical review of mTOR inhibitors and epilepsy: from basic science to clinical trials.Mammalian target of rapamycin (mTOR) pathways in neurological diseases.Influence of miR-155 on Cell Apoptosis in Rats with Ischemic Stroke: Role of the Ras Homolog Enriched in Brain (Rheb)/mTOR Pathway.Regulation of cell death and epileptogenesis by the mammalian target of rapamycin (mTOR): a double-edged sword?Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.Inhibition of mammalian target of rapamycin improves neurobehavioral deficit and modulates immune response after intracerebral hemorrhage in rat.Therapeutic approaches to epileptogenesis--hope on the horizon.Tuberous sclerosis complex as a model disease for developing new therapeutics for epilepsy.Traumatic Brain Injury Stimulates Neural Stem Cell Proliferation via Mammalian Target of Rapamycin Signaling Pathway Activation.Modulators of microglial activation and polarization after intracerebral haemorrhage.TIMP3 Attenuates the Loss of Neural Stem Cells, Mature Neurons and Neurocognitive Dysfunction in Traumatic Brain Injury.Combination therapy targeting Akt and mammalian target of rapamycin improves functional outcome after controlled cortical impact in mice.PI3K-mTOR-S6K Signaling Mediates Neuronal Viability via Collapsin Response Mediator Protein-2 Expression.Sexually dimorphic expression and estradiol mediated up-regulation of a sex-linked ribosomal gene, RPS6, in the zebra finch brain.

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Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury

http://www.wikidata.org/entity/Q28571240

description

2007 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2007 թվականի մայիսին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2007

@ast

im August 2006 veröffentlicher wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2007/05/01)

@sk

vědecký článek publikovaný v roce 2007

@cs

wetenschappelijk artikel (gepubliceerd op 2007/05/01)

@nl

наукова стаття, опублікована в травні 2007

@uk

مقالة علمية نشرت في مايو 2007 حول موضوع: دماغ

@ar

name

Alterations in mammalian targe ...... s after traumatic brain injury

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Alterations in mammalian targe ...... s after traumatic brain injury

@en

Alterations in mammalian targe ...... s after traumatic brain injury

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type

label

Alterations in mammalian targe ...... s after traumatic brain injury

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Alterations in mammalian targe ...... s after traumatic brain injury

@en

Alterations in mammalian targe ...... s after traumatic brain injury

@nl

prefLabel

Alterations in mammalian targe ...... s after traumatic brain injury

@ast

Alterations in mammalian targe ...... s after traumatic brain injury

@en

Alterations in mammalian targe ...... s after traumatic brain injury

@nl

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P356

SJ.JCBFM.9600393

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Journal of Cerebral Blood Flow & Metabolism

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Alterations in mammalian targe ...... s after traumatic brain injury

@en

P2093

Bingren R Hu

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Chunli L Liu

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Ofelia F Alonso

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Shaoyi Chen

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W Dalton Dietrich

http://www.w3.org/2001/XMLSchema#string

P2860

Posttraumatic epilepsy: a major problem in desperate need of major advances

S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling

Insulin activation of Rheb, a mediator of mTOR/S6K/4E-BP signaling, is inhibited by TSC1 and 2

Phosphatidic acid-mediated mitogenic activation of mTOR signaling

Identification of S6 kinase 1 as a novel mammalian target of rapamycin (mTOR)-phosphorylating kinase

Brain-derived neurotrophic factor induces mammalian target of rapamycin-dependent local activation of translation machinery and protein synthesis in neuronal dendrites

Knocking-down the NMDAR1 subunit in a limited amount of neurons in the rat hippocampus impairs learning

Ribosomal protein S6 phosphorylation is a determinant of cell size and glucose homeostasis.

Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E

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10.1038/SJ.JCBFM.9600393

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5

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27

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Coleen M. Atkins

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2006-08-30T00:00:00Z

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6835269

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