Adenovirus E1A inhibits cardiac myocyte-specific gene expression through its amino terminus
about
A novel Rb- and p300-binding protein inhibits transactivation by MyoDThe transcription factors GATA4 and dHAND physically interact to synergistically activate cardiac gene expression through a p300-dependent mechanismSubcellular compartmentalization of E2F family members is required for maintenance of the postmitotic state in terminally differentiated muscleRepression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophyNon-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transitionThe transcriptional co-activators CREB-binding protein (CBP) and p300 play a critical role in cardiac hypertrophy that is dependent on their histone acetyltransferase activityp300 Functions as a coactivator of transcription factor GATA-4.Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.FOG-2 competes with GATA-4 for transcriptional coactivator p300 and represses hypertrophic responses in cardiac myocytes.Transgenic expression in mouse lung reveals distinct biological roles for the adenovirus type 5 E1A 243- and 289-amino-acid proteinsTransforming growth factor beta1 receptor II is downregulated by E1A in adenovirus-infected cellsThe virtue of just enough stress: a molecular model.Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death.The transcriptional co-activators CBP and p300 are activated via phenylephrine through the p42/p44 MAPK cascade.Cell cycle-mediated regulation of smooth muscle alpha-actin gene transcription in fibroblasts and vascular smooth muscle cells involves multiple adenovirus E1A-interacting cofactors.Activation of c-Jun N-terminal kinase promotes survival of cardiac myocytes after oxidative stress.The retinoblastoma gene family members pRB and p107 coactivate the AP-1-dependent mouse tissue factor promoter in fibroblasts.Coordinate inhibition of cytokine-mediated induction of ferritin H, manganese superoxide dismutase, and interleukin-6 by the adenovirus E1A oncogene.
P2860
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P2860
Adenovirus E1A inhibits cardiac myocyte-specific gene expression through its amino terminus
description
1997 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
artículu científicu espublizáu en 1997
@ast
im August 1997 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1997/08/15)
@sk
vědecký článek publikovaný v roce 1997
@cs
wetenschappelijk artikel (gepubliceerd op 1997/08/15)
@nl
наукова стаття, опублікована в серпні 1997
@uk
name
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@ast
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@en
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@nl
type
label
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@ast
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@en
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@nl
prefLabel
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@ast
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@en
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@nl
P2093
P2860
P356
P1476
Adenovirus E1A inhibits cardia ...... ion through its amino terminus
@en
P2093
G. Q. Zeng
K. A. Webster
N. H. Bishopric
P2860
P304
20584–20594
P356
10.1074/JBC.272.33.20584
P407
P577
1997-08-15T00:00:00Z