Increased infarct size and exacerbated apoptosis in the glutathione peroxidase-1 (Gpx-1) knockout mouse brain in response to ischemia/reperfusion injury
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Targeted disruption of SPI3/Serpinb6 does not result in developmental or growth defects, leukocyte dysfunction, or susceptibility to strokeOxidative Stress and the Use of Antioxidants in StrokeSelenoproteins in nervous system development and functionProtein methionine oxidation augments reperfusion injury in acute ischemic strokeImpaired angiogenesis in glutathione peroxidase-1-deficient mice is associated with endothelial progenitor cell dysfunctionDiminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell deathGlutathione peroxidase-1 primes pro-inflammatory cytokine production after LPS challenge in vivo.Ameliorating activity of ginger (Zingiber officinale) extract against lead induced renal toxicity in male rats.Glutathione peroxidase-1 modulates lipopolysaccharide-induced adhesion molecule expression in endothelial cells by altering CD14 expression.Reactive oxygen species enhance insulin sensitivityDifferent strokes for different folks: the rich diversity of animal models of focal cerebral ischemiaMouse Models of Oxidative Stress Indicate a Role for Modulating Healthy Aging.Peripheral nervous system genes expressed in central neurons induce growth on inhibitory substrates.Selenium level and depressive symptoms in a rural elderly Chinese cohort.Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia.MyD88 is a critical regulator of hematopoietic cell-mediated neuroprotection seen after stroke.Histone deacetylase inhibitors prevent oxidative neuronal death independent of expanded polyglutamine repeats via an Sp1-dependent pathwayProteomic analysis of extremely severe hand, foot and mouth disease infected by enterovirus 71Glutathione suppresses cerebral infarct volume and cell death after ischemic injury: involvement of FOXO3 inactivation and Bcl2 expression.An imbalance in antioxidant defense affects cellular function: the pathophysiological consequences of a reduction in antioxidant defense in the glutathione peroxidase-1 (Gpx1) knockout mouse.Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunitiesMitochondrial peroxiredoxin III is a potential target for cancer therapyInhibitor of apoptosis (IAP)-like protein lacks a baculovirus IAP repeat (BIR) domain and attenuates cell death in plant and animal systemsImpact of oxidative stress on neuronal survival.Importance of T lymphocytes in brain injury, immunodeficiency, and recovery after cerebral ischemia.Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.Oxidative stress in atherogenesis and arterial thrombosis: the disconnect between cellular studies and clinical outcomes.Oxidative stress: a pathogenic mechanism for Niemann-Pick type C disease.Neuronal death/survival signaling pathways in cerebral ischemia.Cell-Permeable Peptide Targeting the Nrf2-Keap1 Interaction: A Potential Novel Therapy for Global Cerebral Ischemia.Glutathione efflux and cell death.Apocynin and ebselen reduce influenza A virus-induced lung inflammation in cigarette smoke-exposed mice.Oenanthe Javanica Extract Protects Against Experimentally Induced Ischemic Neuronal Damage via its Antioxidant EffectsAntioxidants in central nervous system diseases: preclinical promise and translational challenges.Mechanisms of stress resistance in Snell dwarf mouse fibroblasts: enhanced antioxidant and DNA base excision repair capacity, but no differences in mitochondrial metabolismOpposite effects of HDAC5 and p300 on MRTF-A-related neuronal apoptosis during ischemia/reperfusion injury in rats.Therapeutic potential of targeting hydrogen peroxide metabolism in the treatment of brain ischaemia.Impact of dietary selenium intake on cardiac health: experimental approaches and human studies.Glutathione peroxidase-1 as a novel therapeutic target for COPD.Revisiting an age-old question regarding oxidative stress.
P2860
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P2860
Increased infarct size and exacerbated apoptosis in the glutathione peroxidase-1 (Gpx-1) knockout mouse brain in response to ischemia/reperfusion injury
description
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2001
@ast
scientific journal article
@en
vedecký článok (publikovaný 2001/09/01)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/09/01)
@nl
наукова стаття, опублікована у вересні 2001
@uk
مقالة علمية نشرت في سبتمبر 2001 حول موضوع: دماغ
@ar
name
Increased infarct size and exa ...... to ischemia/reperfusion injury
@ast
Increased infarct size and exa ...... to ischemia/reperfusion injury
@en
Increased infarct size and exa ...... to ischemia/reperfusion injury
@nl
type
label
Increased infarct size and exa ...... to ischemia/reperfusion injury
@ast
Increased infarct size and exa ...... to ischemia/reperfusion injury
@en
Increased infarct size and exa ...... to ischemia/reperfusion injury
@nl
prefLabel
Increased infarct size and exa ...... to ischemia/reperfusion injury
@ast
Increased infarct size and exa ...... to ischemia/reperfusion injury
@en
Increased infarct size and exa ...... to ischemia/reperfusion injury
@nl
P2093
P921
P1476
Increased infarct size and exa ...... to ischemia/reperfusion injury
@en
P2093
J M Taylor
N J Flentjar
R C Iannello
P304
P356
10.1046/J.1471-4159.2001.00535.X
P407
P577
2001-09-01T00:00:00Z