Diminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell death

Diminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell death

Item
http://www.wikidata.org/entity/Q28591081
Schisandrin B as a hormetic agent for preventing age-related neurodegenerative diseasesAdjunctive dexamethasone affects the expression of genes related to inflammation, neurogenesis and apoptosis in infant rat pneumococcal meningitisImpaired angiogenesis in glutathione peroxidase-1-deficient mice is associated with endothelial progenitor cell dysfunctionRecruitment of BAD by the Chlamydia trachomatis vacuole correlates with host-cell survival.Particular phosphorylation of PI3K/Akt on Thr308 via PDK-1 and PTEN mediates melatonin's neuroprotective activity after focal cerebral ischemia in mice.DJ-1 protects the nigrostriatal axis from the neurotoxin MPTP by modulation of the AKT pathway.Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunitiesDeletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotypeHuman neural stem cells genetically modified to overexpress Akt1 provide neuroprotection and functional improvement in mouse stroke modelAntioxidant gene therapy against neuronal cell death.Allele-specific interaction between glutathione peroxidase 1 and manganese superoxide dismutase affects the levels of Bcl-2, Sirt3 and E-cadherin.Orexin A attenuates palmitic acid-induced hypothalamic cell deathType-I interferons mediate the neuroinflammatory response and neurotoxicity induced by rotenone.Neurotrophic effects of leukemia inhibitory factor on neural cells derived from human embryonic stem cells.Phosphorylation of the translation initiation factor eIF2α at serine 51 determines the cell fate decisions of Akt in response to oxidative stressNeuronal death by oxidative stress involves activation of FOXO3 through a two-arm pathway that activates stress kinases and attenuates insulin-like growth factor I signaling.Baicalein Preventive Treatment Confers Optimal Cardioprotection by PTEN/Akt/NO Activation.
P2860

Diminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell death

Item
http://www.wikidata.org/entity/Q28591081
description
2005 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artikull shkencor
@sq
artículu científicu espublizáu en 2005
@ast
scientific journal article
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в січні 2005
@uk
مقالة علمية (نشرت عام 2005)
@ar
name
Diminished Akt phosphorylation ...... tive stress-induced cell death
@ast
Diminished Akt phosphorylation ...... tive stress-induced cell death
@en
Diminished Akt phosphorylation ...... tive stress-induced cell death
@nl
type
label
Diminished Akt phosphorylation ...... tive stress-induced cell death
@ast
Diminished Akt phosphorylation ...... tive stress-induced cell death
@en
Diminished Akt phosphorylation ...... tive stress-induced cell death
@nl
prefLabel
Diminished Akt phosphorylation ...... tive stress-induced cell death
@ast
Diminished Akt phosphorylation ...... tive stress-induced cell death
@en
Diminished Akt phosphorylation ...... tive stress-induced cell death
@nl
P1433
P1476
P2093
P2860
P304
P31
P3181
P356
P407
P433
P478
P50
P577
P5875
P698
P921
P3181
P356
P1433
P1476
Diminished Akt phosphorylation ...... tive stress-induced cell death
@en
P2093
P2860
P304
P31
P3181
P356
10.1111/J.1471-4159.2004.02863.X
P407
P433
P478
P50
P577
P5875
P698
P921