mTOR and neuronal cell cycle reentry: How impaired brain insulin signaling promotes Alzheimer's disease.
about
Re-imagining Alzheimer's disease - the diminishing importance of amyloid and a glimpse of what lies ahead.Extracellular Tau Oligomers Induce Invasion of Endogenous Tau into the Somatodendritic Compartment and Axonal Transport Dysfunction.Reduced brain insulin signaling: A seminal process in Alzheimer's disease pathogenesis.A unique homeostatic signaling pathway links synaptic inactivity to postsynaptic mTORC1.Amyloid Beta monomers regulate cyclic adenosine monophosphate response element binding protein functions by activating type-1 insulin-like growth factor receptors in neuronal cells.The diabetes drug liraglutide reverses cognitive impairment in mice and attenuates insulin receptor and synaptic pathology in a non-human primate model of Alzheimer's disease.
P2860
mTOR and neuronal cell cycle reentry: How impaired brain insulin signaling promotes Alzheimer's disease.
description
2016 nî lūn-bûn
@nan
2016 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@ast
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@en
type
label
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@ast
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@en
prefLabel
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@ast
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@en
P2093
P2860
P1476
mTOR and neuronal cell cycle r ...... promotes Alzheimer's disease.
@en
P2093
Andrés Norambuena
Antonia Silva
Daniel Baerthlein
Eric Swanson
Erin Kodis
George S Bloom
Horst Wallrabe
Lloyd McMahon
Salvatore Oddo
P2860
P304
P356
10.1016/J.JALZ.2016.08.015
P577
2016-09-29T00:00:00Z