DNA replication stress is a determinant of chronological lifespan in budding yeast. - Wikidata - wikimedia - TriplyDB

DNA replication stress is a determinant of chronological lifespan in budding yeast.

DNA replication stress is a determinant of chronological lifespan in budding yeast.

http://www.wikidata.org/entity/Q30444507

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Replicative and chronological aging in Saccharomyces cerevisiae.The many ways to age for a single yeast cell Aging and cell death in the other yeasts, Schizosaccharomyces pombe and Candida albicans Genome-wide screen in Saccharomyces cerevisiae identifies vacuolar protein sorting, autophagy, biosynthetic, and tRNA methylation genes involved in life span regulation.Growth conditions that increase or decrease lifespan in Saccharomyces cerevisiae lead to corresponding decreases or increases in rates of interstitial deletions and non-reciprocal translocations Down-regulating sphingolipid synthesis increases yeast lifespan Genomic instability is associated with natural life span variation in Saccharomyces cerevisiae.Nucleotide metabolism, oncogene-induced senescence and cancer.Implications of the colonic deposition of free hemoglobin-α chain: a previously unknown tissue by-product in inflammatory bowel disease Nutrient sensing kinases PKA and Sch9 phosphorylate the catalytic domain of the ubiquitin-conjugating enzyme Cdc34.Caloric restriction or catalase inactivation extends yeast chronological lifespan by inducing H2O2 and superoxide dismutase activity.Ammonium is toxic for aging yeast cells, inducing death and shortening of the chronological lifespan Extension of Saccharomyces paradoxus chronological lifespan by retrotransposons in certain media conditions is associated with changes in reactive oxygen species.Growth signaling promotes chronological aging in budding yeast by inducing superoxide anions that inhibit quiescence Regulation of NAD+ metabolism, signaling and compartmentalization in the yeast Saccharomyces cerevisiae DNA replication stress, genome instability and aging.Reactive oxygen species, ageing and the hormesis police.Sub1 and Maf1, two effectors of RNA polymerase III, are involved in the yeast quiescence cycle.Retrotransposition is associated with genome instability during chronological aging.Conserved role of medium acidification in chronological senescence of yeast and mammalian cells An Energy-Independent Pro-longevity Function of Triacylglycerol in Yeast Chronological aging-induced apoptosis in yeast.DNA replication stress-induced loss of reproductive capacity in S. cerevisiae and its inhibition by caloric restriction.Apoptotic death of ageing yeast.Less is more: Nutrient limitation induces cross-talk of nutrient sensing pathways with NAD(+) homeostasis and contributes to longevity.Oncogene homologue Sch9 promotes age-dependent mutations by a superoxide and Rev1/Polzeta-dependent mechanism.Budding yeast SSD1-V regulates transcript levels of many longevity genes and extends chronological life span in purified quiescent cells.Uncoupling reproduction from metabolism extends chronological lifespan in yeast.Chronological aging in Saccharomyces cerevisiae.Growth culture conditions and nutrient signaling modulating yeast chronological longevity.Molecular mechanisms linking the evolutionary conserved TORC1-Sch9 nutrient signalling branch to lifespan regulation in Saccharomyces cerevisiae.Live longer on MARS: a yeast paradigm of mitochondrial adaptive ROS signaling in aging.At neutral pH the chronological lifespan of Hansenula polymorpha increases upon enhancing the carbon source concentrations.Mitochondrial retrograde signaling inhibits the survival during prolong S/G2 arrest in Saccharomyces cerevisiae.Dietary restriction and lifespan: Lessons from invertebrate models.Live fast, die soon: cell cycle progression and lifespan in yeast cells Phosphate-activated cyclin-dependent kinase stabilizes G1 cyclin to trigger cell cycle entry.A set of nutrient limitations trigger yeast cell death in a nitrogen-dependent manner during wine alcoholic fermentation.A new Schizosaccharomyces pombe chronological lifespan assay reveals that caloric restriction promotes efficient cell cycle exit and extends longevity.Loss of histone H3 methylation at lysine 4 triggers apoptosis in Saccharomyces cerevisiae.

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P2860

DNA replication stress is a determinant of chronological lifespan in budding yeast.

http://www.wikidata.org/entity/Q30444507

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2007 nî lūn-bûn

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2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2007 թվականի օգոստոսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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DNA replication stress is a determinant of chronological lifespan in budding yeast.

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Anita Paul

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Daniel L Smith

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Jeffrey S Smith

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Joel A Huberman

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Keshav K Singh

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Li Feng

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Marija Vujcic

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Martin Weinberger

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Robert D Hontz

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William C Burhans

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P2860

Extension of chronological life span in yeast by decreased TOR pathway signaling

Calorie restriction induces mitochondrial biogenesis and bioenergetic efficiency

The WHI1+ gene of Saccharomyces cerevisiae tethers cell division to cell size and is a cyclin homolog

DAF1, a mutant gene affecting size control, pheromone arrest, and cell cycle kinetics of Saccharomyces cerevisiae

Reduced TOR signaling extends chronological life span via increased respiration and upregulation of mitochondrial gene expression

Ras and Gpa2 mediate one branch of a redundant glucose signaling pathway in yeast

Aging: a theory based on free radical and radiation chemistry

Exploring the metabolic and genetic control of gene expression on a genomic scale

Genomic expression programs in the response of yeast cells to environmental changes

Saccharomyces cerevisiae cAMP-dependent protein kinase controls entry into stationary phase through the Rim15p protein kinase.

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