Deafness and permanently reduced potassium channel gene expression and function in hypothyroid Pit1dw mutants.
about
Alström Syndrome protein ALMS1 localizes to basal bodies of cochlear hair cells and regulates cilium-dependent planar cell polarity.Thyroid hormone is required for the pruning of afferent type II spiral ganglion neurons in the mouse cochlea.Potassium ion movement in the inner ear: insights from genetic disease and mouse modelsGenetic background of Prop1(df) mutants provides remarkable protection against hypothyroidism-induced hearing impairment.Specification of skeletal muscle differentiation by repressor element-1 silencing transcription factor (REST)-regulated Kv7.4 potassium channels.A modifier gene alleviates hypothyroidism-induced hearing impairment in Pou1f1dw dwarf mice.The timecourse of apoptotic cell death during postnatal remodeling of the mouse cochlea and its premature onset by triiodothyronine (T3)Spinster homolog 2 (spns2) deficiency causes early onset progressive hearing loss.Developmental and cell-specific expression of thyroid hormone transporters in the mouse cochleaOveractivation of Notch1 signaling induces ectopic hair cells in the mouse inner ear in an age-dependent manner.Age-dependent in vivo conversion of mouse cochlear pillar and Deiters' cells to immature hair cells by Atoh1 ectopic expressionThyroid hormone receptor-α and vascular function.Age-Related Hearing Loss and Degeneration of Cochlear Hair Cells in Mice Lacking Thyroid Hormone Receptor β1Thyroid hormone is required for pruning, functioning and long-term maintenance of afferent inner hair cell synapses.Vertical transmission of hypopituitarism: critical importance of appropriate interpretation of thyroid function tests and levothyroxine therapy during pregnancy.Hearing impairment in hypothyroid dwarf mice caused by mutations of the thyroid peroxidase geneMaking sense with thyroid hormone--the role of T(3) in auditory development.Hearing Loss and Otopathology Following Systemic and Intracerebroventricular Delivery of 2-Hydroxypropyl-Beta-Cyclodextrin.Cochlear dysfunction in patients with acute hypothyroidism.Cyclodextrins and Iatrogenic Hearing Loss: New Drugs with Significant Risk.Deletion of the Thyroid Hormone-Activating Type 2 Deiodinase Rescues Cone Photoreceptor Degeneration but Not Deafness in Mice Lacking Type 3 Deiodinase.Deafness and loss of cochlear hair cells in the absence of thyroid hormone transporters Slc16a2 (Mct8) and Slc16a10 (Mct10).GC-B Deficient Mice With Axon Bifurcation Loss Exhibit Compromised Auditory ProcessingGrxcr2 is required for stereocilia morphogenesis in the cochlea
P2860
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P2860
Deafness and permanently reduced potassium channel gene expression and function in hypothyroid Pit1dw mutants.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@ast
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@en
type
label
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@ast
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@en
prefLabel
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@ast
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants.
@en
P2093
P2860
P1476
Deafness and permanently reduc ...... in hypothyroid Pit1dw mutants
@en
P2093
David F Dolan
Mirna Mustapha
Tzy-Wen Gong
Yehoash Raphael
P2860
P304
P356
10.1523/JNEUROSCI.4957-08.2009
P407
P577
2009-01-01T00:00:00Z