Supporting cell characteristics in long-deafened aged mouse ears
about
Co-administration of cisplatin and furosemide causes rapid and massive loss of cochlear hair cells in mice.Fractalkine Signaling Regulates Macrophage Recruitment into the Cochlea and Promotes the Survival of Spiral Ganglion Neurons after Selective Hair Cell LesionSelective deletion of cochlear hair cells causes rapid age-dependent changes in spiral ganglion and cochlear nucleus neuronsSystemic lipopolysaccharide induces cochlear inflammation and exacerbates the synergistic ototoxicity of kanamycin and furosemide.Aminoglycoside ototoxicity and hair cell ablation in the adult gerbil: A simple model to study hair cell loss and regeneration.Making sense of Wnt signaling-linking hair cell regeneration to development.A novel role of cytosolic protein synthesis inhibition in aminoglycoside ototoxicity.Changes in the adult vertebrate auditory sensory epithelium after trauma.Postnatal development, maturation and aging in the mouse cochlea and their effects on hair cell regeneration.Notch signaling limits supporting cell plasticity in the hair cell-damaged early postnatal murine cochleaSelective ablation of pillar and deiters' cells severely affects cochlear postnatal development and hearing in mice.Defining the cellular environment in the organ of Corti following extensive hair cell loss: a basis for future sensory cell replacement in the Cochlea.Adeno-associated virus-mediated gene delivery into the scala media of the normal and deafened adult mouse ear.In vivo proliferation of postmitotic cochlear supporting cells by acute ablation of the retinoblastoma protein in neonatal miceDevelopment of hair cells in inner ear is associated with expression and promoter methylation of Notch-1 in postnatal mice.Cdc42-dependent structural development of auditory supporting cells is required for wound healing at adulthood.Non-autonomous Cellular Responses to Ototoxic Drug-Induced Stress and Death.Genetic disruption of fractalkine signaling leads to enhanced loss of cochlear afferents following ototoxic or acoustic injury.Epithelial-mesenchymal transition, and collective and individual cell migration regulate epithelial changes in the amikacin-damaged organ of Corti.Expression of acetylated tubulin in the postnatal developing mouse cochlea
P2860
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P2860
Supporting cell characteristics in long-deafened aged mouse ears
description
2009 nî lūn-bûn
@nan
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Supporting cell characteristics in long-deafened aged mouse ears
@ast
Supporting cell characteristics in long-deafened aged mouse ears
@en
type
label
Supporting cell characteristics in long-deafened aged mouse ears
@ast
Supporting cell characteristics in long-deafened aged mouse ears
@en
prefLabel
Supporting cell characteristics in long-deafened aged mouse ears
@ast
Supporting cell characteristics in long-deafened aged mouse ears
@en
P2860
P1433
P1476
Supporting cell characteristics in long-deafened aged mouse ears
@en
P2093
Elizabeth C Oesterle
Sean Campbell
P2860
P2888
P304
P356
10.1007/S10162-009-0183-X
P577
2009-07-31T00:00:00Z
P5875
P6179
1044060118