The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy. - Wikidata - wikimedia - TriplyDB

The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy.

The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy.

http://www.wikidata.org/entity/Q30494710

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Epithelial cell polarity: a major gatekeeper against cancer?Epithelial homeostasis Cell competition: how to eliminate your neighbours Transgenic expression of the Helicobacter pylori virulence factor CagA promotes apoptosis or tumorigenesis through JNK activation in Drosophila Multiple strategies of oxygen supply in Drosophila malignancies identify tracheogenesis as a novel cancer hallmark dMyc functions downstream of Yorkie to promote the supercompetitive behavior of hippo pathway mutant cells.Drosophila insulin and target of rapamycin (TOR) pathways regulate GSK3 beta activity to control Myc stability and determine Myc expression in vivo A tumor-suppressing mechanism in Drosophila involving cell competition and the Hippo pathway.Regulation of Hippo signaling by Jun kinase signaling during compensatory cell proliferation and regeneration, and in neoplastic tumors.The Ecdysone receptor constrains wingless expression to pattern cell cycle across the Drosophila wing margin in a Cyclin B-dependent manner.Drosophila Myc: A master regulator of cellular performance.Identification of novel Ras-cooperating oncogenes in Drosophila melanogaster: a RhoGEF/Rho-family/JNK pathway is a central driver of tumorigenesis.Steep differences in wingless signaling trigger Myc-independent competitive cell interactions.Identification of cis-Regulatory Elements in the dmyc Gene of Drosophila Melanogaster.Tumor suppression by cell competition through regulation of the Hippo pathway.Epithelial Tumors Originate in Tumor Hotspots, a Tissue-Intrinsic Microenvironment.MYC, Cell Competition, and Cell Death in Cancer: The Inseparable Triad.Mechanisms of cell competition: themes and variations.Epithelial neoplasia in Drosophila entails switch to primitive cell states Cell wars: regulation of cell survival and proliferation by cell competition Cell competition and its implications for development and cancer.Troponin-I enhances and is required for oncogenic overgrowth Socializing with MYC: cell competition in development and as a model for premalignant cancer.The dark side of fly TNF: an ancient developmental proof reading mechanism turned into tumor promoter.Impact of interactions between normal and transformed epithelial cells and the relevance to cancer.Drosophila cancer models.New frontiers in cell competition.The Scribble-Dlg-Lgl polarity module in development and cancer: from flies to man.Inference of Cell Mechanics in Heterogeneous Epithelial Tissue Based on Multivariate Clone Shape Quantification.Scribble acts in the Drosophila fat-hippo pathway to regulate warts activity.Differences in levels of the transmembrane protein Crumbs can influence cell survival at clonal boundaries.Human Cancer Cells Signal Their Competitive Fitness Through MYC Activity Development of an optimized synthetic Notch receptor as an in vivo cell-cell contact sensor.How winner cells cause the demise of loser cells: cell competition causes apoptosis of suboptimal cells: their dregs are removed by hemocytes, thus preserving tissue homeostasis.Selector genes display tumor cooperation and inhibition in Drosophila epithelium in a developmental context-dependent manner.miR-9a mediates the role of Lethal giant larvae as an epithelial growth inhibitor in Drosophila.The ecdysone receptor signalling regulates microvilli formation in follicular epithelial cells.Changing mutational and adaptive landscapes and the genesis of cancer.Modelling Cooperative Tumorigenesis in Drosophila.Drosophila as a Model System to Study Nonautonomous Mechanisms Affecting Tumour Growth and Cell Death.

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The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy.

http://www.wikidata.org/entity/Q30494710

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

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name

The lethal giant larvae tumour ...... to promote clonal malignancy.

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The lethal giant larvae tumour ...... to promote clonal malignancy.

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The lethal giant larvae tumour ...... to promote clonal malignancy.

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The lethal giant larvae tumour ...... to promote clonal malignancy.

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The lethal giant larvae tumour ...... to promote clonal malignancy.

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The lethal giant larvae tumour ...... to promote clonal malignancy.

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Dennis Strand

http://www.w3.org/2001/XMLSchema#string

Flavio Garoia

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Francesca Froldi

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Marcello Ziosi

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Nicola A Grzeschik

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P2860

The Hallmarks of Cancer

Dlg, Scribble and Lgl in cell polarity, cell proliferation and cancer

Deregulation of scribble promotes mammary tumorigenesis and reveals a role for cell polarity in carcinoma

Putting tumours in context

Myc deletion rescues Apc deficiency in the small intestine

Epithelial-mesenchymal transitions in development and pathologies

Analysis of genetic mosaics in developing and adult Drosophila tissues

aPKCzeta cortical loading is associated with Lgl cytoplasmic release and tumor growth in Drosophila and human epithelia.

Scribble mutants promote aPKC and JNK-dependent epithelial neoplasia independently of Crumbs.

Control of wing size and proportions by Drosophila myc

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scholarly article

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10.1186/1741-7007-8-33

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8

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Helena E. Richardson

Annalisa Pession

Daniela Grifoni

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2010-04-07T00:00:00Z

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43072599

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1049887351

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20374622

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2877678

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