Anthrax lethal toxin induced lysosomal membrane permeabilization and cytosolic cathepsin release is Nlrp1b/Nalp1b-dependent.
about
Role of the inflammasome, IL-1β, and IL-18 in bacterial infectionsAnthrax Toxins in Context of Bacillus anthracis Spores and Spore GerminationThe endolysosomal system in cell death and survivalAnthrax lethal toxin activates the inflammasome in sensitive rat macrophagesCellular adaptation to anthrax lethal toxin-induced mitochondrial cholesterol enrichment, hyperpolarization, and reactive oxygen species generation through downregulating MLN64 in macrophagesAuranofin protects against anthrax lethal toxin-induced activation of the Nlrp1b inflammasomeProteasome inhibitors prevent caspase-1-mediated disease in rodents challenged with anthrax lethal toxin.Programmed cell death and its role in inflammationAllelic variation on murine chromosome 11 modifies host inflammatory responses and resistance to Bacillus anthracisProteolytic processing of Nlrp1b is required for inflammasome activity.Selective inhibitor of endosomal trafficking pathways exploited by multiple toxins and viruses.Inhibition of the Plasma-Membrane-Associated Serine Protease Cathepsin G by Mycobacterium tuberculosis Rv3364c Suppresses Caspase-1 and Pyroptosis in Macrophages.Anthrax and the inflammasomeDistinct cathepsins control necrotic cell death mediated by pyroptosis inducers and lysosome-destabilizing agents.Anthrax lethal factor activates K(+) channels to induce IL-1β secretion in macrophages.Role of lysosome rupture in controlling Nlrp3 signaling and necrotic cell death.Cytosolic flagellin-induced lysosomal pathway regulates inflammasome-dependent and -independent macrophage responsesIntracellular sensing of microbes and danger signals by the inflammasomes.Anthrax lethal toxin and the induction of CD4 T cell immunity.Mechanisms of NOD-like receptor-associated inflammasome activation.E3 ubiquitin ligases Pellinos as regulators of pattern recognition receptor signaling and immune responses.Caspase-1 protein induces apoptosis-associated speck-like protein containing a caspase recruitment domain (ASC)-mediated necrosis independently of its catalytic activity.Oleanolic acid arrests cell cycle and induces apoptosis via ROS-mediated mitochondrial depolarization and lysosomal membrane permeabilization in human pancreatic cancer cells.Leukotoxin (Leukothera®) targets active leukocyte function antigen-1 (LFA-1) protein and triggers a lysosomal mediated cell death pathway.The NLRP12 inflammasome recognizes Yersinia pestis.The acidic transformed nano-VO2 causes macrophage cell death by the induction of lysosomal membrane permeabilization and Ca2+ efflux.
P2860
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P2860
Anthrax lethal toxin induced lysosomal membrane permeabilization and cytosolic cathepsin release is Nlrp1b/Nalp1b-dependent.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@ast
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@en
type
label
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@ast
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@en
prefLabel
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@ast
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@en
P2093
P2860
P1433
P1476
Anthrax lethal toxin induced l ...... se is Nlrp1b/Nalp1b-dependent.
@en
P2093
Joseph A Loo
Kathleen M Averette
Kenneth A Bradley
Matthew R Pratt
Sara Bassilian
Tom W Muir
Yanan Yang
P2860
P356
10.1371/JOURNAL.PONE.0007913
P407
P577
2009-11-18T00:00:00Z