Hypoxia is important in the biology and aggression of human glial brain tumors. - Wikidata - wikimedia - TriplyDB

Hypoxia is important in the biology and aggression of human glial brain tumors.

Hypoxia is important in the biology and aggression of human glial brain tumors.

http://www.wikidata.org/entity/Q31137856

about

Mitochondrial metabolic remodeling in response to genetic and environmental perturbations Glioblastoma Stem Cells Microenvironment: The Paracrine Roles of the Niche in Drug and Radioresistance Stress Response Leading to Resistance in Glioblastoma-The Need for Innovative Radiotherapy (iRT) Concepts Heparin in malignant glioma: review of preclinical studies and clinical results Sustained radiosensitization of hypoxic glioma cells after oxygen pretreatment in an animal model of glioblastoma and in vitro models of tumor hypoxia.Immunotherapy of Malignant Tumors in the Brain: How Different from Other Sites?Exosomes reflect the hypoxic status of glioma cells and mediate hypoxia-dependent activation of vascular cells during tumor development CD133 is a marker of bioenergetic stress in human glioma Recapitulation of tumor heterogeneity and molecular signatures in a 3D brain cancer model with decreased sensitivity to histone deacetylase inhibition Glioma cells suppress hypoxia-induced endothelial cell apoptosis and promote the angiogenic process GAPDH is not regulated in human glioblastoma under hypoxic conditions.Interleukin-1β promotes hypoxia-induced apoptosis of glioblastoma cells by inhibiting hypoxia-inducible factor-1 mediated adrenomedullin production Clinical biomarkers for hypoxia targeting In vivo profiling of hypoxic gene expression in gliomas using the hypoxia marker EF5 and laser-capture microdissection.Hypoxia increases the expression of stem-cell markers and promotes clonogenicity in glioblastoma neurospheres Hypoxic signature of microRNAs in glioblastoma: insights from small RNA deep sequencing.Protein phosphatase 2A mediates dormancy of glioblastoma multiforme-derived tumor stem-like cells during hypoxia.An ANOCEF genomic and transcriptomic microarray study of the response to radiotherapy or to alkylating first-line chemotherapy in glioblastoma patients.Biodistribution and dosimetry of (18)F-EF5 in cancer patients with preliminary comparison of (18)F-EF5 uptake versus EF5 binding in human glioblastoma Differential glioma-associated tumor antigen expression profiles of human glioma cells grown in hypoxia.Superior efficacy of tumor cell vaccines grown in physiologic oxygen Hypoxia triggers a proangiogenic pathway involving cancer cell microvesicles and PAR-2-mediated heparin-binding EGF signaling in endothelial cells.HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells γ₁34.5-deleted HSV-1-expressing human cytomegalovirus IRS1 gene kills human glioblastoma cells as efficiently as wild-type HSV-1 in normoxia or hypoxia.Very high levels of lactate dehydrogenase at diagnosis predict central nervous system relapse in acute promyelocytic leukaemia Regional hypoxia in glioblastoma multiforme quantified with [18F]fluoromisonidazole positron emission tomography before radiotherapy: correlation with time to progression and survival Prospective cohort study of radiotherapy with concomitant and adjuvant temozolomide chemotherapy for glioblastoma patients with no or minimal residual enhancing tumor load after surgery A Lab Assembled Microcontroller-Based Sensor Module for Continuous Oxygen Measurement in Portable Hypoxia Chambers.Hypoxia Moderates γ(1)34.5-Deleted Herpes Simplex Virus Oncolytic Activity in Human Glioma Xenoline Primary Cultures.Glioma revisited: from neurogenesis and cancer stem cells to the epigenetic regulation of the niche pH, Lactate, and Hypoxia: Reciprocity in Regulating High-Affinity Monocarboxylate Transporter Expression in Glioblastoma A simplified synthesis of the hypoxia imaging agent 2-(2-Nitro-1H-imidazol-1-yl)-N-(2,2,3,3,3-[(18)F]pentafluoropropyl)-acetamide ([18F]EF5)Cancer as a moving target: understanding the composition and rebound growth kinetics of recurrent tumors.Hypoxia induces macrophage polarization and re-education toward an M2 phenotype in U87 and U251 glioblastoma models Pediatric glioma stem cells: biologic strategies for oncolytic HSV virotherapy In vitro expansion of human glioblastoma cells at non-physiological oxygen tension irreversibly alters subsequent in vivo aggressiveness and AC133 expression.MicroRNA-584-3p, a novel tumor suppressor and prognostic marker, reduces the migration and invasion of human glioma cells by targeting hypoxia-induced ROCK1.BH3 mimetics reactivate autophagic cell death in anoxia-resistant malignant glioma cells.Reoxygenation of hypoxic glioblastoma multiforme cells potentiates the killing effect of an interleukin-13-based cytotoxin.Cytotoxicity of withaferin A in glioblastomas involves induction of an oxidative stress-mediated heat shock response while altering Akt/mTOR and MAPK signaling pathways.

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P2860

Hypoxia is important in the biology and aggression of human glial brain tumors.

http://www.wikidata.org/entity/Q31137856

description

2004 nî lūn-bûn

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2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Hypoxia is important in the biology and aggression of human glial brain tumors.

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Hypoxia is important in the biology and aggression of human glial brain tumors.

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type

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Hypoxia is important in the biology and aggression of human glial brain tumors.

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Hypoxia is important in the biology and aggression of human glial brain tumors.

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prefLabel

Hypoxia is important in the biology and aggression of human glial brain tumors.

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Hypoxia is important in the biology and aggression of human glial brain tumors.

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1078-0432.CCR-04-1081

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Clinical Cancer Research

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Hypoxia is important in the biology and aggression of human glial brain tumors

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Cameron J Koch

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Deirdre P Magarelli

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Isolde Dunphy

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Kevin D Judy

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Kevin Jenkins

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M Sean Grady

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Sydney M. Evans

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