HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells - Wikidata - wikimedia - TriplyDB

HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells

HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells

http://www.wikidata.org/entity/Q35249827

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PI3K/AKT/mTOR Pathway in Angiogenesis A conceptually new treatment approach for relapsed glioblastoma: coordinated undermining of survival paths with nine repurposed drugs (CUSP9) by the International Initiative for Accelerated Improvement of Glioblastoma Care Novel delivery strategies for glioblastoma Nelfinavir and other protease inhibitors in cancer: mechanisms involved in anticancer activity A novel inhibitor of hypoxia-inducible factor-1α P3155 also modulates PI3K pathway and inhibits growth of prostate cancer cells Neoplasia: Where We Have Been and Where We Are Going Neoplasia: the second decade.Alterations in the Notch4 pathway in cerebral endothelial cells by the HIV aspartyl protease inhibitor, nelfinavir.Radiosensitization of epidermal growth factor receptor/HER2-positive pancreatic cancer is mediated by inhibition of Akt independent of ras mutational status.Insights into the broad cellular effects of nelfinavir and the HIV protease inhibitors supporting their role in cancer treatment and prevention Tumor vascular changes mediated by inhibition of oncogenic signaling.The HIV protease inhibitor, nelfinavir, as a novel therapeutic approach for the treatment of refractory pediatric leukemia.Recent advances in drug repositioning for the discovery of new anticancer drugs Markers of inflammation and coagulation indicate a prothrombotic state in HIV-infected patients with long-term use of antiretroviral therapy with or without abacavir.Tamoxifen enhances the cytotoxic effects of nelfinavir in breast cancer cells.A meta-analysis of HIF-1α and esophageal squamous cell carcinoma (ESCC) risk.Advances in the targeted therapy of liposarcoma.The HIV protease inhibitor nelfinavir inhibits Kaposi's sarcoma-associated herpesvirus replication in vitro Control of proliferation in astrocytoma cells by the receptor tyrosine kinase/PI3K/AKT signaling axis and the use of PI-103 and TCN as potential anti-astrocytoma therapies.Breast Cancer in the Setting of HIV Impact of Human Immunodeficiency Virus in the Pathogenesis and Outcome of Patients with Glioblastoma Multiforme.Targeting the hypoxia-inducible factor (HIF) pathway in cancer.Modulating the tumor microenvironment to increase radiation responsiveness.Combining metformin and nelfinavir exhibits synergistic effects against the growth of human cervical cancer cells and xenograft in nude mice.Targeting the AKT pathway: Repositioning HIV protease inhibitors as radiosensitizers.The influence of SRPK1 on glioma apoptosis, metastasis, and angiogenesis through the PI3K/Akt signaling pathway under normoxia.Knockdown of the AKT3 (PKBγ), PI3KCA, and VEGFR2 genes by RNA interference suppresses glioblastoma multiforme T98G cells invasiveness in vitro.HIV-protease inhibitors for the treatment of cancer: Repositioning HIV protease inhibitors while developing more potent NO-hybridized derivatives?Amprenavir inhibits the migration in human hepatocarcinoma cell and the growth of xenografts.Glioblastoma Multiforme in an HIV-Infected Patient: An Unexpected Diagnosis.Lopinavir inhibits meningioma cell proliferation by Akt independent mechanism.Neoplasia: An Anniversary of Progress.HIF-1α- Targeting Acriflavine Provides Long Term Survival and Radiological Tumor Response in Brain Cancer Therapy.Phase II trial of ritonavir/lopinavir in patients with progressive or recurrent high-grade gliomas.Effect of vascular endothelial growth factors A, C, and D in HIV-associated pre-eclampsia.The Impact of Human Papilloma Viruses, Matrix Metallo-Proteinases and HIV Protease Inhibitors on the Onset and Progression of Uterine Cervix Epithelial Tumors: A Review of Preclinical and Clinical Studies.Prevalence of human immunodeficiency virus infection in brain glioma patients: Is the virus protective from gliomas?HIV antiretroviral exposure in pregnancy induces detrimental placenta vascular changes that are rescued by progesterone supplementation.Anticancer and Differentiation Properties of the Nitric Oxide Derivative of Lopinavir in Human Glioblastoma Cells

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P2860

HIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cells

http://www.wikidata.org/entity/Q35249827

description

2006 nî lūn-bûn

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2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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type

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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HIV protease inhibitors decrea ...... ogenesis in glioblastoma cells

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Amit Maity

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Anjali K Gupta

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George J Cerniglia

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Nabendu Pore

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P2860

Regulation of hypoxia-inducible factor 1alpha is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway

Hypoxia-inducible factor determines sensitivity to inhibitors of mTOR in kidney cancer.

Bevacizumab plus irinotecan, fluorouracil, and leucovorin for metastatic colorectal cancer

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

Expression of hypoxia-inducible factor 1alpha in brain tumors: association with angiogenesis, invasion, and progression

Loss of PTEN facilitates HIF-1-mediated gene expression

Hypoxia inducible factor-1alpha as a cancer drug target

Effect of VEGF receptor inhibitor PTK787/ZK222584 [correction of ZK222548] combined with ionizing radiation on endothelial cells and tumour growth

Hypoxia-inducible factor 1alpha (HIF-1alpha) protein is rapidly degraded by the ubiquitin-proteasome system under normoxic conditions. Its stabilization by hypoxia depends on redox-induced changes

Modulation of hypoxia-inducible factor 1alpha expression by the epidermal growth factor/phosphatidylinositol 3-kinase/PTEN/AKT/FRAP pathway in human prostate cancer cells: implications for tumor angiogenesis and therapeutics

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10.1593/NEO.06535

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11

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8

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