Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.
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Cardiac Na+ current regulation by pyridine nucleotidesProtein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondriaCyclosporin A corrects mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathiesThe human silent information regulator (Sir)2 homologue hSIRT3 is a mitochondrial nicotinamide adenine dinucleotide-dependent deacetylaseModulation of the matrix redox signaling by mitochondrial Ca(2.)Germline energetics, aging, and female infertilityPostconditioning signalling in the heart: mechanisms and translatabilityStructural characterization of a human cytosolic NMN/NaMN adenylyltransferase and implication in human NAD biosynthesisSIRT3 in Cardiac Physiology and DiseaseIntramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPTCa2+-dependent permeability transition regulation in rat brain mitochondria by 2',3'-cyclic nucleotides and 2',3'-cyclic nucleotide 3'-phosphodiesteraseMitochondrial reactive oxygen species production in excitable cells: modulators of mitochondrial and cell function.Effect of transient and permanent permeability transition pore opening on NAD(P)H localization in intact cells.Regulation of the mitochondrial permeability transition in kidney proximal tubules and its alteration during hypoxia-reoxygenationRegulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).Hydrogen sulfide alleviates cardiac contractile dysfunction in an Akt2-knockout murine model of insulin resistance: role of mitochondrial injury and apoptosis.Mechanisms of ER Stress-Mediated Mitochondrial Membrane Permeabilization.Mitochondrial dysfunction induced by nuclear poly(ADP-ribose) polymerase-1: a treatable cause of cell death in stroke.Oxidative stress: Biomarkers and novel therapeutic pathways.New strategies for Alzheimer's disease and cognitive impairment.Protein kinase C epsilon regulates mitochondrial pools of Nampt and NAD following resveratrol and ischemic preconditioning in the rat cortexDiabetes mellitus: channeling care through cellular discoverymTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.NAD+ depletion is necessary and sufficient for poly(ADP-ribose) polymerase-1-mediated neuronal deathVincristine attenuates N-methyl-N'-nitro-N-nitrosoguanidine-induced poly-(ADP) ribose polymerase activity in cardiomyocytes.Mitochondrial dysfunction in the limelight of Parkinson's disease pathogenesis.Protective effects of early hypoxic post-conditioning in cultured cortical neurons.Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction.Novel O-palmitolylated beta-E1 subunit of pyruvate dehydrogenase is phosphorylated during ischemia/reperfusion injuryThe effects of idebenone on mitochondrial bioenergeticsCalcium and mitochondrial reactive oxygen species generation: how to read the facts.Reactive oxygen species originating from mitochondria regulate the cardiac sodium channel.Novel therapeutic strategies for ischemic heart diseaseCardiac mitochondria and arrhythmias.Mitochondrial dysfunction and defective autophagy in the pathogenesis of collagen VI muscular dystrophies.Low-intensity aerobic interval training attenuates pathological left ventricular remodeling and mitochondrial dysfunction in aortic-banded miniature swineMelanocytes from Patients Affected by Ullrich Congenital Muscular Dystrophy and Bethlem Myopathy have Dysfunctional Mitochondria That Can be Rescued with Cyclophilin Inhibitors.Novel avenues of drug discovery and biomarkers for diabetes mellitus.Interaction of TPPP/p25 protein with glyceraldehyde-3-phosphate dehydrogenase and their co-localization in Lewy bodies.The role of mitochondria in protection of the heart by preconditioning.
P2860
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P2860
Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.
description
2000 nî lūn-bûn
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2000 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
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2000 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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name
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@ast
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@en
type
label
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@ast
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@en
prefLabel
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@ast
Opening of the mitochondrial p ...... emic reperfusion of the heart.
@en
P50
P356
P1476
Opening of the mitochondrial p ...... hemic reperfusion of the heart
@en
P2093
P304
P356
10.1074/JBC.M006825200
P407
P577
2000-11-09T00:00:00Z