Selective activation of mitogen-activated protein (MAP) kinase kinase 3 and p38alpha MAP kinase is essential for cyclic AMP-dependent UCP1 expression in adipocytes.
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Behavioral and cerebellar transmission deficits in mice lacking the autism-linked gene islet brain-2Sestrin2, a Regulator of Thermogenesis and Mitohormesis in Brown Adipose TissueBerardinelli-seip congenital lipodystrophy 2/seipin is a cell-autonomous regulator of lipolysis essential for adipocyte differentiationβ-Adrenergic stimulation does not activate p38 MAP kinase or induce PGC-1α in skeletal muscleEpigallocatechin-3-gallate (EGCG), a green tea polyphenol, suppresses hepatic gluconeogenesis through 5'-AMP-activated protein kinase.Post-translational regulation of Oct4 transcriptional activityp38 Mitogen-activated protein kinase plays a stimulatory role in hepatic gluconeogenesis.Liver X receptor alpha is a transcriptional repressor of the uncoupling protein 1 gene and the brown fat phenotype.Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species.Prostaglandin E2 inhibits α-smooth muscle actin transcription during myofibroblast differentiation via distinct mechanisms of modulation of serum response factor and myocardin-related transcription factor-ACyclic AMP stimulates Mrp2 translocation by activating p38{alpha} MAPK in hepatic cells.Syndecan-1 is required to maintain intradermal fat and prevent cold stress.Cyclic AMP inhibits p38 activation via CREB-induced dynein light chain.INTRACELLULAR SIGNALING BY BILE ACIDS.A subcutaneous adipose tissue-liver signalling axis controls hepatic gluconeogenesis.PEST motif serine and tyrosine phosphorylation controls vascular endothelial growth factor receptor 2 stability and downregulation.Glucagon stimulates hepatic FGF21 secretion through a PKA- and EPAC-dependent posttranscriptional mechanism.Hypoxia-induced inflammatory cytokine secretion in human adipose tissue stromovascular cells.Cellular Aging Contributes to Failure of Cold-Induced Beige Adipocyte Formation in Old Mice and Humans.Phospholipase C-related Catalytically Inactive Protein Is a New Modulator of Thermogenesis Promoted by β-Adrenergic Receptors in Brown AdipocytesASK1 signalling regulates brown and beige adipocyte function.MAP kinases and the control of nuclear events.Activation of mTORC1 is essential for β-adrenergic stimulation of adipose browning.Inactivation of UCP1 and the glycerol phosphate cycle synergistically increases energy expenditure to resist diet-induced obesity.p38 MAPK α and β isoforms differentially regulate plasma membrane localization of MRP2.MAPK signaling pathways in the regulation of hematopoiesis.PGC-1 coactivators in cardiac development and diseaseβ-Adrenoceptor Signaling Networks in Adipocytes for Recruiting Stored Fat and Energy Expenditure.Brown and beige fat: the metabolic function, induction, and therapeutic potential.New therapeutic approaches for the treatment of obesity.The role of innate immunity in the regulation of brown and beige adipogenesis.miRNA-32 Drives Brown Fat Thermogenesis and Trans-activates Subcutaneous White Fat Browning in Mice.Cannabinoid receptor stimulation impairs mitochondrial biogenesis in mouse white adipose tissue, muscle, and liver: the role of eNOS, p38 MAPK, and AMPK pathways.Follistatin Targets Distinct Pathways To Promote Brown Adipocyte Characteristics in Brown and White Adipose Tissues.Induction of uncoupling protein-1 in mouse embryonic fibroblast-derived adipocytes by retinoic acid.p38α MAPK antagonizing JNK to control the hepatic fat accumulation in pediatric patients onset intestinal failure.Dietary menthol-induced TRPM8 activation enhances WAT "browning" and ameliorates diet-induced obesity.GSK3 is a negative regulator of the thermogenic program in brown adipocytes.Pycnogenol® Induces Browning of White Adipose Tissue through the PKA Signaling Pathway in Apolipoprotein E-Deficient Mice.p38 Mitogen-activated protein kinase mediates free fatty acid-induced gluconeogenesis in hepatocytes.
P2860
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P2860
Selective activation of mitogen-activated protein (MAP) kinase kinase 3 and p38alpha MAP kinase is essential for cyclic AMP-dependent UCP1 expression in adipocytes.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@ast
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@en
type
label
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@ast
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@en
prefLabel
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@ast
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@en
P2093
P2860
P1476
Selective activation of mitoge ...... UCP1 expression in adipocytes.
@en
P2093
Fatiha Moukdar
Jacques Robidoux
Kiefer W Daniel
Lisa M Floering
Sheila Collins
Wenhong Cao
P2860
P304
P356
10.1128/MCB.25.13.5466-5479.2005
P407
P577
2005-07-01T00:00:00Z